Yasuha Arai
Kyoto, Japan
Last Updated: 09/26/2026
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Found 23 publications
Intra-patient neuraminidase mutations in avian H5N1 influenza virus reduce sialidase activity to complement weaker hemagglutinin binding and facilitate human infection.
Journal: PLoS pathogens
Published: July 09, 2025
The host tropism of current zoonotic H7N9 viruses depends mainly on an acid-labile hemagglutinin with a single amino acid mutation in the stalk region.
Journal: PLoS pathogens
Published: November 09, 2022
ACE2 N-glycosylation modulates interactions with SARS-CoV-2 spike protein in a site-specific manner.
ACE2 N-glycosylation modulates interactions with SARS-CoV-2 spike protein in a site-specific manner.
Journal: Communications biology
Published: July 10, 2022
Double mutations in the H9N2 avian influenza virus PB2 gene act cooperatively to increase viral host adaptation and replication for human infections.
Journal: The Journal of general virology
Published: June 01, 2021
Development of genus-specific universal primers for the detection of flaviviruses.
Journal: Virology journal
Published: April 14, 2021
"Genetic tuning" of avian influenza virus host adaptation from birds to humans.
Journal: Biosafety and health
Published: September 23, 2020
PA Mutations Inherited during Viral Evolution Act Cooperatively To Increase Replication of Contemporary H5N1 Influenza Virus with an Expanded Host Range.
Journal: Journal of virology
Published: August 12, 2020
H9N2 Influenza Virus Infections in Human Cells Require a Balance between Neuraminidase Sialidase Activity and Hemagglutinin Receptor Affinity.
Journal: Journal of virology
Published: June 26, 2020
Infection of Human Tracheal Epithelial Cells by H5 Avian Influenza Virus Is Regulated by the Acid Stability of Hemagglutinin and the pH of Target Cell Endosomes.
Journal: Viruses
Published: December 24, 2019
Last Updated: 09/26/2026