Yilei Meng

Shanghai, 200120, China

Last Updated: 09/26/2026

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Found 8 publications

LncRNA Foxo6os as a Novel " Scaffold" Mediates MYBPC3 in Combating Pathological Cardiac Hypertrophy and Heart Failure.

LncRNA Foxo6os as a Novel " Scaffold" Mediates MYBPC3 in Combating Pathological Cardiac Hypertrophy and Heart Failure.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: April 24, 2025

LPItabformer: Enhancing generalization in predicting lncRNA-protein interactions via a tabular Transformer.

LPItabformer: Enhancing generalization in predicting lncRNA-protein interactions via a tabular Transformer.

Journal: Computational and structural biotechnology journal

Published: January 31, 2025

Unmasking Protein Phosphatase 2A Regulatory Subunit B as a Crucial Factor in the Progression of Dilated Cardiomyopathy.

Unmasking Protein Phosphatase 2A Regulatory Subunit B as a Crucial Factor in the Progression of Dilated Cardiomyopathy.

Journal: Biomedicines

Published: July 23, 2024

Kcnh2 deletion is associated with rat embryonic development defects via destruction of KCNH2‑integrin β1 complex.

Kcnh2 deletion is associated with rat embryonic development defects via destruction of KCNH2‑integrin β1 complex.

Journal: International journal of molecular medicine

Published: May 18, 2023

Natural Tissue-Imprinted Biointerface for the Topographical Education of a Biomimetic Cell Sheet.

Natural Tissue-Imprinted Biointerface for the Topographical Education of a Biomimetic Cell Sheet.

Journal: Langmuir : the ACS journal of surfaces and colloids

Published: June 22, 2022

Kcnh2 mediates FAK/AKT-FOXO3A pathway to attenuate sepsis-induced cardiac dysfunction.

Kcnh2 mediates FAK/AKT-FOXO3A pathway to attenuate sepsis-induced cardiac dysfunction.

Journal: Cell proliferation

Published: August 19, 2020

ERG1 plays an essential role in rat cardiomyocyte fate decision by mediating AKT signaling.

ERG1 plays an essential role in rat cardiomyocyte fate decision by mediating AKT signaling.

Journal: Stem cells (Dayton, Ohio)

Published: July 19, 2020

miR-29b-3p protects cardiomyocytes against endotoxin-induced apoptosis and inflammatory response through targeting FOXO3A.

miR-29b-3p protects cardiomyocytes against endotoxin-induced apoptosis and inflammatory response through targeting FOXO3A.

Journal: Cellular signalling

Published: March 01, 2020

Showing 1-8 of 8

Last Updated: 09/26/2026