Yinan F. Luo

71 Xinmin Avenue
Changchun, 130021, China

Last Updated: 09/26/2026

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Found 24 publications

Corrigendum to "RSL3 induced autophagic death in glioma cells via causing glycolysis dysfunction" [Biochem. Biophys. Res. Commun. 518 (2019) 590-597].

Corrigendum to "RSL3 induced autophagic death in glioma cells via causing glycolysis dysfunction" [Biochem. Biophys. Res. Commun. 518 (2019) 590-597].

Journal: Biochemical and biophysical research communications

Published: September 04, 2023

Incidence of unintended pregnancy within 2 years after delivery and its influencing factors in China

Incidence of unintended pregnancy within 2 years after delivery and its influencing factors in China

Journal: Zhonghua fu chan ke za zhi

Published: September 22, 2021

Transcription factor ZNF703 activates linc-UBC1 to stimulate the progression of glioma.

Transcription factor ZNF703 activates linc-UBC1 to stimulate the progression of glioma.

Journal: European review for medical and pharmacological sciences

Published: April 10, 2020

Corrigendum to "MLKL contributes to shikonin-induced glioma cell necroptosis via promotion of chromatinolysis" [Canc. Lett. 467 (2019) 58-71].

Corrigendum to "MLKL contributes to shikonin-induced glioma cell necroptosis via promotion of chromatinolysis" [Canc. Lett. 467 (2019) 58-71].

Journal: Cancer letters

Published: January 10, 2020

RSL3 induced autophagic death in glioma cells via causing glycolysis dysfunction.

RSL3 induced autophagic death in glioma cells via causing glycolysis dysfunction.

Journal: Biochemical and biophysical research communications

Published: August 01, 2019

MLKL contributes to shikonin-induced glioma cell necroptosis via promotion of chromatinolysis.

MLKL contributes to shikonin-induced glioma cell necroptosis via promotion of chromatinolysis.

Journal: Cancer letters

Published: July 15, 2019

Pseudolaric acid B triggers ferroptosis in glioma cells via activation of Nox4 and inhibition of xCT.

Pseudolaric acid B triggers ferroptosis in glioma cells via activation of Nox4 and inhibition of xCT.

Journal: Cancer letters

Published: March 15, 2018

RIP1 and RIP3 contribute to shikonin-induced glycolysis suppression in glioma cells via increase of intracellular hydrogen peroxide.

RIP1 and RIP3 contribute to shikonin-induced glycolysis suppression in glioma cells via increase of intracellular hydrogen peroxide.

Journal: Cancer letters

Published: February 10, 2018

RIP1 and RIP3 contribute to shikonin-induced DNA double-strand breaks in glioma cells via increase of intracellular reactive oxygen species.

RIP1 and RIP3 contribute to shikonin-induced DNA double-strand breaks in glioma cells via increase of intracellular reactive oxygen species.

Journal: Cancer letters

Published: October 08, 2016

Trehalose Inhibits Protein Aggregation Caused by Transient Ischemic Insults Through Preservation of Proteasome Activity, Not via Induction of Autophagy.

Trehalose Inhibits Protein Aggregation Caused by Transient Ischemic Insults Through Preservation of Proteasome Activity, Not via Induction of Autophagy.

Journal: Molecular neurobiology

Published: April 13, 2016

JNK Activation Contributes to Oxidative Stress-Induced Parthanatos in Glioma Cells via Increase of Intracellular ROS Production.

JNK Activation Contributes to Oxidative Stress-Induced Parthanatos in Glioma Cells via Increase of Intracellular ROS Production.

Journal: Molecular neurobiology

Published: January 19, 2016

Pristimerin triggers AIF-dependent programmed necrosis in glioma cells via activation of JNK.

Pristimerin triggers AIF-dependent programmed necrosis in glioma cells via activation of JNK.

Journal: Cancer letters

Published: November 20, 2015

Showing 1-12 of 24

Last Updated: 09/26/2026