Yingwei Li

107 Wenhua Xi Road
Jinan, China

Last Updated: 09/26/2026

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Found 40 publications

Correction: The splicing factor WBP11 mediates MCM7 intron retention to promote the malignant progression of ovarian cancer.

Correction: The splicing factor WBP11 mediates MCM7 intron retention to promote the malignant progression of ovarian cancer.

Journal: Oncogene
Published: April 23, 2026

Disruption of the SNRPF-DDX24-E2F4 Feedback Loop Uncouples Splicing and Transcriptional Regulation to Suppress Ovarian Cancer Progression.

Disruption of the SNRPF-DDX24-E2F4 Feedback Loop Uncouples Splicing and Transcriptional Regulation to Suppress Ovarian Cancer Progression.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: November 18, 2025

SNRPD2-CPSF7-UBE2K axis drives ovarian cancer progression via alternative splicing-polyadenylation crosstalk.

SNRPD2-CPSF7-UBE2K axis drives ovarian cancer progression via alternative splicing-polyadenylation crosstalk.

Journal: Oncogene
Published: November 04, 2025

Spliceosomal component SNRPE drives cell proliferation by regulating CTP synthase 1 mRNA splicing in ovarian cancer.

Spliceosomal component SNRPE drives cell proliferation by regulating CTP synthase 1 mRNA splicing in ovarian cancer.

Journal: Oncogene
Published: August 23, 2025

SNRPD2-mediated regulation of DDX39B splicing promotes endometrial cancer progression by suppressing the activation of CTSC cryptic exons.

SNRPD2-mediated regulation of DDX39B splicing promotes endometrial cancer progression by suppressing the activation of CTSC cryptic exons.

Journal: Cell death & disease
Published: July 17, 2025

Spliceosomal GTPase EFTUD2 mediates DDX41 intron retention to promote the malignant progression of ovarian cancer.

Spliceosomal GTPase EFTUD2 mediates DDX41 intron retention to promote the malignant progression of ovarian cancer.

Journal: British journal of cancer
Published: January 02, 2025

SNRPB-mediated regulation of DDX39A splicing promotes ovarian cancer progression by regulating α6 integrin subunit expression.

SNRPB-mediated regulation of DDX39A splicing promotes ovarian cancer progression by regulating α6 integrin subunit expression.

Journal: Oncogene
Published: November 07, 2024

Targeting the splicing factor SNRPB inhibits endometrial cancer progression by retaining the POLD1 intron.

Targeting the splicing factor SNRPB inhibits endometrial cancer progression by retaining the POLD1 intron.

Journal: Experimental & molecular medicine
Published: July 15, 2024

The splicing factor WBP11 mediates MCM7 intron retention to promote the malignant progression of ovarian cancer.

The splicing factor WBP11 mediates MCM7 intron retention to promote the malignant progression of ovarian cancer.

Journal: Oncogene
Published: August 25, 2023

Inhibition of SF3B1 improves the immune microenvironment through pyroptosis and synergizes with αPDL1 in ovarian cancer.

Inhibition of SF3B1 improves the immune microenvironment through pyroptosis and synergizes with αPDL1 in ovarian cancer.

Journal: Cell death & disease
Published: June 09, 2023

The splicing factor SNRPB promotes ovarian cancer progression through regulating aberrant exon skipping of POLA1 and BRCA2.

The splicing factor SNRPB promotes ovarian cancer progression through regulating aberrant exon skipping of POLA1 and BRCA2.

Journal: Oncogene
Published: February 05, 2023

Oncolytic adenovirus with MUC16-BiTE shows enhanced antitumor immune response by reversing the tumor microenvironment in PDX model of ovarian cancer.

Oncolytic adenovirus with MUC16-BiTE shows enhanced antitumor immune response by reversing the tumor microenvironment in PDX model of ovarian cancer.

Journal: Oncoimmunology
Published: July 08, 2022
Showing 1-12 of 40

Last Updated: 09/26/2026