Yllza Jasiqi
Lausanne, VD 1015, Switzerland
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 9 publications
Modeling Parkinson's pathology in human iPSC dopaminergic neurons uncovers key mechanisms of Lewy body formation and heterogeneity.
Journal: Science advances
Published: July 10, 2026
Differential role of C-terminal truncations on alpha-synuclein pathology and Lewy body formation.
Journal: NPJ Parkinson's disease
Published: November 10, 2024
Author Correction: O-GlcNAc forces an α-synuclein amyloid strain with notably diminished seeding and pathology.
Journal: Nature chemical biology
Published: February 27, 2024
Recombinant Full-Length TDP-43 Oligomers Retain Their Ability to Bind RNAs, Are Not Toxic, and Do Not Seed TDP-43 Aggregation in Vitro.
Journal: ACS chemical neuroscience
Published: December 20, 2023
Effective Inhibition of TDP-43 Aggregation by Native State Stabilization.
Journal: Angewandte Chemie (International ed. in English)
Published: October 09, 2023
O-GlcNAc modification forces the formation of an α-Synuclein amyloid-strain with notably diminished seeding activity and pathology.
Journal: bioRxiv : the preprint server for biology
Published: March 22, 2023
O-GlcNAc forces an α-synuclein amyloid strain with notably diminished seeding and pathology.
Journal: Nature chemical biology
Published: March 13, 2023
Revisiting the specificity and ability of phospho-S129 antibodies to capture alpha-synuclein biochemical and pathological diversity.
Journal: NPJ Parkinson's disease
Published: March 29, 2022
The UPR sensor IRE1α and the adenovirus E3-19K glycoprotein sustain persistent and lytic infections.
The UPR sensor IRE1α and the adenovirus E3-19K glycoprotein sustain persistent and lytic infections.
Journal: Nature communications
Published: January 14, 2020
Showing 1-9 of 9
Last Updated: 09/26/2026