Yohko Kage

Miyazaki, 8891692, Japan

Last Updated: 09/26/2026

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Found 11 publications

The formin Fhod3 is required for sarcomere reassembly after mitosis in postnatal cardiomyocytes.

The formin Fhod3 is required for sarcomere reassembly after mitosis in postnatal cardiomyocytes.

Journal: Cellular and molecular life sciences : CMLS
Published: January 05, 2026

Structural basis underlying the autoinhibition of the formin FHOD1 and its phosphorylation-dependent activation.

Structural basis underlying the autoinhibition of the formin FHOD1 and its phosphorylation-dependent activation.

Journal: The Journal of biological chemistry
Published: July 24, 2025

Reversible neuropathic pain model created by long-term optogenetic nociceptor stimulation using light-responsive pain mice.

Reversible neuropathic pain model created by long-term optogenetic nociceptor stimulation using light-responsive pain mice.

Journal: PloS one
Published: January 02, 2025

The expression of the formin Fhod3 in mouse tongue striated muscle.

The expression of the formin Fhod3 in mouse tongue striated muscle.

Journal: Cell structure and function
Published: October 09, 2024

Selective optogenetic activation of NaV1.7-expressing afferents in NaV1.7-ChR2 mice induces nocifensive behavior without affecting responses to mechanical and thermal stimuli.

Selective optogenetic activation of NaV1.7-expressing afferents in NaV1.7-ChR2 mice induces nocifensive behavior without affecting responses to mechanical and thermal stimuli.

Journal: PloS one
Published: June 11, 2022

Extracellular signal-regulated kinase phosphorylation enhancement and NaV1.7 sodium channel upregulation in rat dorsal root ganglia neurons contribute to resiniferatoxin-induced neuropathic pain: The efficacy and mechanism of pulsed radiofrequency therapy.

Extracellular signal-regulated kinase phosphorylation enhancement and NaV1.7 sodium channel upregulation in rat dorsal root ganglia neurons contribute to resiniferatoxin-induced neuropathic pain: The efficacy and mechanism of pulsed radiofrequency therapy.

Journal: Molecular pain
Published: April 14, 2022

Fhod3 Controls the Dendritic Spine Morphology of Specific Subpopulations of Pyramidal Neurons in the Mouse Cerebral Cortex.

Fhod3 Controls the Dendritic Spine Morphology of Specific Subpopulations of Pyramidal Neurons in the Mouse Cerebral Cortex.

Journal: Cerebral cortex (New York, N.Y. : 1991)
Published: December 10, 2019

Fhod1, an actin-organizing formin family protein, is dispensable for cardiac development and function in mice.

Fhod1, an actin-organizing formin family protein, is dispensable for cardiac development and function in mice.

Journal: Cytoskeleton (Hoboken, N.J.)
Published: January 18, 2019

Interaction between cardiac myosin-binding protein C and formin Fhod3.

Interaction between cardiac myosin-binding protein C and formin Fhod3.

Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: April 25, 2018

The actin-organizing formin protein Fhod3 is required for postnatal development and functional maintenance of the adult heart in mice.

The actin-organizing formin protein Fhod3 is required for postnatal development and functional maintenance of the adult heart in mice.

Journal: The Journal of biological chemistry
Published: August 23, 2017

Transgenic Expression of the Formin Protein Fhod3 Selectively in the Embryonic Heart: Role of Actin-Binding Activity of Fhod3 and Its Sarcomeric Localization during Myofibrillogenesis.

Transgenic Expression of the Formin Protein Fhod3 Selectively in the Embryonic Heart: Role of Actin-Binding Activity of Fhod3 and Its Sarcomeric Localization during Myofibrillogenesis.

Journal: PloS one
Published: August 29, 2015
Showing 1-11 of 11

Last Updated: 09/26/2026