Yong K. Han

Daegu, Republic of Korea

Last Updated: 09/26/2026

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Found 10 publications

Hepatic injury-induced remote kidney injury is associated with impaired hepatic clearance of cell-free hemoglobin.

Hepatic injury-induced remote kidney injury is associated with impaired hepatic clearance of cell-free hemoglobin.

Journal: Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
Published: August 25, 2026

Cystathionine γ-lyase deficiency exacerbates high-fat diet-induced kidney and liver injury associated with disrupted glutathione homeostasis.

Cystathionine γ-lyase deficiency exacerbates high-fat diet-induced kidney and liver injury associated with disrupted glutathione homeostasis.

Journal: Free radical biology & medicine
Published: April 08, 2026

Diarylpropionitrile, a selective agonist of estrogen receptor β, protects the kidney against ischemia-reperfusion injury and enhances the repair of tubular epithelial cells.

Diarylpropionitrile, a selective agonist of estrogen receptor β, protects the kidney against ischemia-reperfusion injury and enhances the repair of tubular epithelial cells.

Journal: Naunyn-Schmiedeberg's archives of pharmacology
Published: March 04, 2026

Deletion of exocyst component 5 suppresses repair of injured kidney by limiting cell proliferation.

Deletion of exocyst component 5 suppresses repair of injured kidney by limiting cell proliferation.

Journal: Cell death discovery
Published: January 20, 2026

Cystathionine γ-Lyase Protects Against Choline-Deficient High-Fat Diet-Induced Metabolic Dysfunction-Associated Steatotic Liver Disease Through the Cysteine-Glutathione Axis in Mice.

Cystathionine γ-Lyase Protects Against Choline-Deficient High-Fat Diet-Induced Metabolic Dysfunction-Associated Steatotic Liver Disease Through the Cysteine-Glutathione Axis in Mice.

Journal: Antioxidants & redox signaling
Published: September 17, 2025

Deficiency of exocyst complex component Exoc5 exacerbates the progression of kidney fibrosis.

Deficiency of exocyst complex component Exoc5 exacerbates the progression of kidney fibrosis.

Journal: Experimental & molecular medicine
Published: May 14, 2025

Kidney ischemia/reperfusion injury causes cholangiocytes primary cilia disruption and abnormal bile secretion.

Kidney ischemia/reperfusion injury causes cholangiocytes primary cilia disruption and abnormal bile secretion.

Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: February 29, 2024

Short-term control of diet affects cisplatin-induced acute kidney injury through modulation of mitochondrial dynamics and mitochondrial GSH.

Short-term control of diet affects cisplatin-induced acute kidney injury through modulation of mitochondrial dynamics and mitochondrial GSH.

Journal: Physiological reports
Published: May 18, 2022

Cisplatin induces lung cell cilia disruption and lung damage via oxidative stress.

Cisplatin induces lung cell cilia disruption and lung damage via oxidative stress.

Journal: Free radical biology & medicine
Published: October 19, 2021

Oxidative stress following acute kidney injury causes disruption of lung cell cilia and their release into the bronchoaveolar lavage fluid and lung injury, which are exacerbated by Idh2 deletion.

Oxidative stress following acute kidney injury causes disruption of lung cell cilia and their release into the bronchoaveolar lavage fluid and lung injury, which are exacerbated by Idh2 deletion.

Journal: Redox biology
Published: July 01, 2021
Showing 1-10 of 10

Last Updated: 09/26/2026