Yongcheng Pan
Key Laboratory Of Hunan Province In Neurodegenerative Disorders
Changsha, China
Changsha, China
Last Updated: 09/26/2026
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Found 27 publications
PolyG Fibrils Coalesce Into Nuclear Ribbons That Engage Proteostasis Machinery in Neuronal Intranuclear Inclusion Disease.
Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: February 28, 2026
Modeling neuronal intranuclear inclusion disease: A review of animal and human-derived cellular models and mechanistic insights.
Journal: Zoological research
Published: December 01, 2025
HnRNP M expression rescues neurodegeneration in neuronal intranuclear inclusion disease mouse model by restoring dysregulated RNA splicing and transcription.
Journal: Cell & bioscience
Published: July 07, 2025
Expression of expanded GGC repeats within NOTCH2NLC causes cardiac dysfunction in mouse models.
Journal: Cell & bioscience
Published: August 01, 2023
Rhes depletion promotes striatal accumulation and aggregation of mutant huntingtin in a presymptomatic HD mouse model.
Journal: Frontiers in aging neuroscience
Published: June 08, 2023
GGC expansions in NOTCH2NLC contribute to Parkinson disease and dopaminergic neuron degeneration.
Journal: European journal of neurology
Published: May 27, 2023
Expression of expanded GGC repeats within NOTCH2NLC causes behavioral deficits and neurodegeneration in a mouse model of neuronal intranuclear inclusion disease.
Journal: Science advances
Published: November 23, 2022
Identification of a Novel Heterozygous Mutation in the EIF2B4 Gene Associated With Vanishing White Matter Disease.
Journal: Frontiers in bioengineering and biotechnology
Published: March 22, 2022
Last Updated: 09/26/2026