Yoojung Kwon

Chuncheon, Republic of Korea

Last Updated: 09/26/2026

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Found 12 publications

HDAC6-MYCN-CXCL3 axis mediates allergic inflammation and is necessary for allergic inflammation-promoted cellular interactions.

HDAC6-MYCN-CXCL3 axis mediates allergic inflammation and is necessary for allergic inflammation-promoted cellular interactions.

Journal: Molecular immunology
Published: September 01, 2023

HDAC6 and CXCL13 Mediate Atopic Dermatitis by Regulating Cellular Interactions and Expression Levels of miR-9 and SIRT1.

HDAC6 and CXCL13 Mediate Atopic Dermatitis by Regulating Cellular Interactions and Expression Levels of miR-9 and SIRT1.

Journal: Frontiers in pharmacology
Published: April 06, 2021

EGR3-HDAC6-IL-27 Axis Mediates Allergic Inflammation and Is Necessary for Tumorigenic Potential of Cancer Cells Enhanced by Allergic Inflammation-Promoted Cellular Interactions.

EGR3-HDAC6-IL-27 Axis Mediates Allergic Inflammation and Is Necessary for Tumorigenic Potential of Cancer Cells Enhanced by Allergic Inflammation-Promoted Cellular Interactions.

Journal: Frontiers in immunology
Published: March 14, 2021

MiR-154-5p-MCP1 Axis Regulates Allergic Inflammation by Mediating Cellular Interactions.

MiR-154-5p-MCP1 Axis Regulates Allergic Inflammation by Mediating Cellular Interactions.

Journal: Frontiers in immunology
Published: February 03, 2021

Homoharringtonine Inhibits Allergic Inflammations by Regulating NF-κB-miR-183-5p-BTG1 Axis.

Homoharringtonine Inhibits Allergic Inflammations by Regulating NF-κB-miR-183-5p-BTG1 Axis.

Journal: Frontiers in pharmacology
Published: March 02, 2020

Exosomal MicroRNAs as Mediators of Cellular Interactions Between Cancer Cells and Macrophages.

Exosomal MicroRNAs as Mediators of Cellular Interactions Between Cancer Cells and Macrophages.

Journal: Frontiers in immunology
Published: January 31, 2020

FcεRI-HDAC3-MCP1 Signaling Axis Promotes Passive Anaphylaxis Mediated by Cellular Interactions.

FcεRI-HDAC3-MCP1 Signaling Axis Promotes Passive Anaphylaxis Mediated by Cellular Interactions.

Journal: International journal of molecular sciences
Published: September 03, 2019

Targeting Autophagy for Overcoming Resistance to Anti-EGFR Treatments.

Targeting Autophagy for Overcoming Resistance to Anti-EGFR Treatments.

Journal: Cancers
Published: July 29, 2019

MiR-135-5p-p62 Axis Regulates Autophagic Flux, Tumorigenic Potential, and Cellular Interactions Mediated by Extracellular Vesicles During Allergic Inflammation.

MiR-135-5p-p62 Axis Regulates Autophagic Flux, Tumorigenic Potential, and Cellular Interactions Mediated by Extracellular Vesicles During Allergic Inflammation.

Journal: Frontiers in immunology
Published: December 11, 2018

Role of HDAC3-miRNA-CAGE Network in Anti-Cancer Drug-Resistance.

Role of HDAC3-miRNA-CAGE Network in Anti-Cancer Drug-Resistance.

Journal: International journal of molecular sciences
Published: November 27, 2018

Human Adipose Tissue-Derived Mesenchymal Stem Cells Attenuate Atopic Dermatitis by Regulating the Expression of MIP-2, miR-122a-SOCS1 Axis, and Th1/Th2 Responses.

Human Adipose Tissue-Derived Mesenchymal Stem Cells Attenuate Atopic Dermatitis by Regulating the Expression of MIP-2, miR-122a-SOCS1 Axis, and Th1/Th2 Responses.

Journal: Frontiers in pharmacology
Published: May 22, 2018

MicroRNA-26a/-26b-COX-2-MIP-2 Loop Regulates Allergic Inflammation and Allergic Inflammation-promoted Enhanced Tumorigenic and Metastatic Potential of Cancer Cells.

MicroRNA-26a/-26b-COX-2-MIP-2 Loop Regulates Allergic Inflammation and Allergic Inflammation-promoted Enhanced Tumorigenic and Metastatic Potential of Cancer Cells.

Journal: The Journal of biological chemistry
Published: February 12, 2015
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Last Updated: 09/26/2026