Yoshinobu Ichimura
Tokyo, Japan
Last Updated: 09/26/2026
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Found 24 publications
A redox-independent stress response mediated by phase-separated SQSTM1/p62.
Journal: Autophagy reports
Published: May 21, 2025
Phosphorylation of phase-separated p62 bodies by ULK1 activates a redox-independent stress response.
Phosphorylation of phase-separated p62 bodies by ULK1 activates a redox-independent stress response.
Journal: The EMBO journal
Published: December 20, 2022
Considering the mechanism by which droplets of ALS-FTD-associated SQSTM1/p62 mutants cause pathology.
Journal: Autophagy reports
Published: March 17, 2022
Phase-separated protein droplets of amyotrophic lateral sclerosis-associated p62/SQSTM1 mutants show reduced inner fluidity.
Journal: The Journal of biological chemistry
Published: June 07, 2021
A description of novel variants and review of phenotypic spectrum in UBA5-related early epileptic encephalopathy.
Journal: Cold Spring Harbor molecular case studies
Published: September 28, 2020
p62/SQSTM1-droplet serves as a platform for autophagosome formation and anti-oxidative stress response.
Journal: Nature communications
Published: May 18, 2020
Inhibitors of the protein-protein interaction between phosphorylated p62 and Keap1 attenuate chemoresistance in a human hepatocellular carcinoma cell line.
Journal: Free radical research
Published: February 21, 2020
Autophagy regulates lipid metabolism through selective turnover of NCoR1.
Journal: Nature communications
Published: May 27, 2018
Activation of p62/SQSTM1-Keap1-Nuclear Factor Erythroid 2-Related Factor 2 Pathway in Cancer.
Journal: Frontiers in oncology
Published: March 22, 2018
Biallelic UFM1 and UFC1 mutations expand the essential role of ufmylation in brain development.
Journal: Brain : a journal of neurology
Published: December 25, 2017
Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.
Journal: Molecular and cellular biology
Published: December 12, 2017
Last Updated: 09/26/2026