Youfen Lin

Fuzhou, 350005, China

Last Updated: 09/26/2026

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Found 8 publications

Corrigendum to "Suppression of TLR4 prevents diabetic bone loss by regulating FTO-mediated m6A modification" [Int. Immunopharmacol. 122 (2023) 110510].

Corrigendum to "Suppression of TLR4 prevents diabetic bone loss by regulating FTO-mediated m6A modification" [Int. Immunopharmacol. 122 (2023) 110510].

Journal: International immunopharmacology

Published: December 12, 2024

Author Correction: Suppression of IRF9 Promotes Osteoclast Differentiation by Decreased Ferroptosis via STAT3 Activation.

Author Correction: Suppression of IRF9 Promotes Osteoclast Differentiation by Decreased Ferroptosis via STAT3 Activation.

Journal: Inflammation

Published: March 15, 2024

The AKT inhibitor AZD5363 elicits synthetic lethality in ARID1A-deficient gastric cancer cells via induction of pyroptosis.

The AKT inhibitor AZD5363 elicits synthetic lethality in ARID1A-deficient gastric cancer cells via induction of pyroptosis.

Journal: British journal of cancer

Published: July 03, 2023

Suppression of IRF9 Promotes Osteoclast Differentiation by Decreased Ferroptosis via STAT3 Activation.

Suppression of IRF9 Promotes Osteoclast Differentiation by Decreased Ferroptosis via STAT3 Activation.

Journal: Inflammation

Published: April 04, 2023

Suppression of TLR4 prevents diabetic bone loss by regulating FTO-mediated m6A modification.

Suppression of TLR4 prevents diabetic bone loss by regulating FTO-mediated m6A modification.

Journal: International immunopharmacology

Published: April 02, 2023

Glycolipid toxicity induces osteogenic dysfunction via the TLR4/S100B pathway.

Glycolipid toxicity induces osteogenic dysfunction via the TLR4/S100B pathway.

Journal: International immunopharmacology

Published: February 16, 2021

Chromatin remodelling factor BAF155 protects hepatitis B virus X protein (HBx) from ubiquitin-independent proteasomal degradation.

Chromatin remodelling factor BAF155 protects hepatitis B virus X protein (HBx) from ubiquitin-independent proteasomal degradation.

Journal: Emerging microbes & infections

Published: September 20, 2019

TLR4 knockout can improve dysfunction of β-cell by rebalancing proteomics disorders in pancreas of obese rats.

TLR4 knockout can improve dysfunction of β-cell by rebalancing proteomics disorders in pancreas of obese rats.

Journal: Endocrine

Published: May 04, 2019

Showing 1-8 of 8

Last Updated: 09/26/2026