Youhou Kang

Toronto, ON, Canada

Last Updated: 09/26/2026

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Found 18 publications

Septin5 deletion enhances β-cell exocytosis by releasing microtubule-tethered insulin granules onto plasma membrane.

Septin5 deletion enhances β-cell exocytosis by releasing microtubule-tethered insulin granules onto plasma membrane.

Journal: Nature communications
Published: February 22, 2024

Plasma membrane flipping of Syntaxin-2 regulates its inhibitory action on insulin granule exocytosis.

Plasma membrane flipping of Syntaxin-2 regulates its inhibitory action on insulin granule exocytosis.

Journal: Nature communications
Published: February 07, 2022

Pancreas-specific SNAP23 depletion prevents pancreatitis by attenuating pathological basolateral exocytosis and formation of trypsin-activating autolysosomes.

Pancreas-specific SNAP23 depletion prevents pancreatitis by attenuating pathological basolateral exocytosis and formation of trypsin-activating autolysosomes.

Journal: Autophagy
Published: November 20, 2020

SNAP23 depletion enables more SNAP25/calcium channel excitosome formation to increase insulin exocytosis in type 2 diabetes.

SNAP23 depletion enables more SNAP25/calcium channel excitosome formation to increase insulin exocytosis in type 2 diabetes.

Journal: JCI insight
Published: April 19, 2019

Kv2.1 clusters on β-cell plasma membrane act as reservoirs that replenish pools of newcomer insulin granule through their interaction with syntaxin-3.

Kv2.1 clusters on β-cell plasma membrane act as reservoirs that replenish pools of newcomer insulin granule through their interaction with syntaxin-3.

Journal: The Journal of biological chemistry
Published: March 04, 2018

Pancreatitis-Induced Depletion of Syntaxin 2 Promotes Autophagy and Increases Basolateral Exocytosis.

Pancreatitis-Induced Depletion of Syntaxin 2 Promotes Autophagy and Increases Basolateral Exocytosis.

Journal: Gastroenterology
Published: May 23, 2017

New Roles of Syntaxin-1A in Insulin Granule Exocytosis and Replenishment.

New Roles of Syntaxin-1A in Insulin Granule Exocytosis and Replenishment.

Journal: The Journal of biological chemistry
Published: November 26, 2016

Munc18b Increases Insulin Granule Fusion, Restoring Deficient Insulin Secretion in Type-2 Diabetes Human and Goto-Kakizaki Rat Islets with Improvement in Glucose Homeostasis.

Munc18b Increases Insulin Granule Fusion, Restoring Deficient Insulin Secretion in Type-2 Diabetes Human and Goto-Kakizaki Rat Islets with Improvement in Glucose Homeostasis.

Journal: EBioMedicine
Published: June 13, 2016

Syntaxin 2 Acts as Inhibitory SNARE for Insulin Granule Exocytosis.

Syntaxin 2 Acts as Inhibitory SNARE for Insulin Granule Exocytosis.

Journal: Diabetes
Published: May 18, 2016

Synaptotagmin-7 Functions to Replenish Insulin Granules for Exocytosis in Human Islet β-Cells.

Synaptotagmin-7 Functions to Replenish Insulin Granules for Exocytosis in Human Islet β-Cells.

Journal: Diabetes
Published: October 19, 2015

Syntaxin-3 Binds and Regulates Both R- and L-Type Calcium Channels in Insulin-Secreting INS-1 832/13 Cells.

Syntaxin-3 Binds and Regulates Both R- and L-Type Calcium Channels in Insulin-Secreting INS-1 832/13 Cells.

Journal: PloS one
Published: October 14, 2015

Munc18c mediates exocytosis of pre-docked and newcomer insulin granules underlying biphasic glucose stimulated insulin secretion in human pancreatic beta-cells.

Munc18c mediates exocytosis of pre-docked and newcomer insulin granules underlying biphasic glucose stimulated insulin secretion in human pancreatic beta-cells.

Journal: Molecular metabolism
Published: January 21, 2015
Showing 1-12 of 18

Last Updated: 09/26/2026