Young K. Im

Montreal, QC, Canada

Last Updated: 09/26/2026

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Found 9 publications

Complex I inhibition combined with TLR activation in the breast tumor microenvironment educates cytotoxic neutrophils.

Complex I inhibition combined with TLR activation in the breast tumor microenvironment educates cytotoxic neutrophils.

Journal: Science advances

Published: July 11, 2025

A second-generation eIF4A RNA helicase inhibitor exploits translational reprogramming as a vulnerability in triple-negative breast cancer.

A second-generation eIF4A RNA helicase inhibitor exploits translational reprogramming as a vulnerability in triple-negative breast cancer.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: January 17, 2024

STAT1 potentiates oxidative stress revealing a targetable vulnerability that increases phenformin efficacy in breast cancer.

STAT1 potentiates oxidative stress revealing a targetable vulnerability that increases phenformin efficacy in breast cancer.

Journal: Nature communications

Published: July 01, 2020

Interplay between ShcA Signaling and PGC-1α Triggers Targetable Metabolic Vulnerabilities in Breast Cancer.

Interplay between ShcA Signaling and PGC-1α Triggers Targetable Metabolic Vulnerabilities in Breast Cancer.

Journal: Cancer research

Published: November 30, 2017

Integration of Distinct ShcA Signaling Complexes Promotes Breast Tumor Growth and Tyrosine Kinase Inhibitor Resistance.

Integration of Distinct ShcA Signaling Complexes Promotes Breast Tumor Growth and Tyrosine Kinase Inhibitor Resistance.

Journal: Molecular cancer research : MCR

Published: October 25, 2017

Translational and HIF-1α-Dependent Metabolic Reprogramming Underpin Metabolic Plasticity and Responses to Kinase Inhibitors and Biguanides.

Translational and HIF-1α-Dependent Metabolic Reprogramming Underpin Metabolic Plasticity and Responses to Kinase Inhibitors and Biguanides.

Journal: Cell metabolism

Published: July 06, 2017

The Shc1 adaptor simultaneously balances Stat1 and Stat3 activity to promote breast cancer immune suppression.

The Shc1 adaptor simultaneously balances Stat1 and Stat3 activity to promote breast cancer immune suppression.

Journal: Nature communications

Published: July 05, 2016

The ShcA adaptor activates AKT signaling to potentiate breast tumor angiogenesis by stimulating VEGF mRNA translation in a 4E-BP-dependent manner.

The ShcA adaptor activates AKT signaling to potentiate breast tumor angiogenesis by stimulating VEGF mRNA translation in a 4E-BP-dependent manner.

Journal: Oncogene

Published: September 21, 2013

The ShcA PTB domain functions as a biological sensor of phosphotyrosine signaling during breast cancer progression.

The ShcA PTB domain functions as a biological sensor of phosphotyrosine signaling during breast cancer progression.

Journal: Cancer research

Published: May 23, 2013

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Last Updated: 09/26/2026