Yu Kawakami
Nagoya, 4668550, Japan
Last Updated: 09/26/2026
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Found 10 publications
Neuronal TDP-43 regulates myelin formation via neurexin 1 mRNA stabilization.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: February 25, 2026
The TDP-43I383V heterozygous mutation results in increased TDP-43 expression and altered neuronal activity in ALS patient-derived iPSC motor neurons.
Journal: Neuroscience research
Published: October 08, 2025
Downregulation of NEAT1 due to loss of TDP-43 function exacerbates motor neuron degeneration in amyotrophic lateral sclerosis.
Journal: Brain communications
Published: February 05, 2025
Truncation mutation of CHMP2B disrupts late endosome function but reduces TDP-43 aggregation through HSP70 upregulation.
Journal: Neurochemistry international
Published: January 12, 2025
IκB kinase phosphorylates cytoplasmic TDP-43 and promotes its proteasome degradation.
Journal: The Journal of cell biology
Published: February 19, 2023
DEFENSIVE Stroke Scale: Novel Diagnostic Tool for Predicting Posterior Circulation Infarction in the Emergency Department.
Journal: Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association
Published: November 26, 2018
Collagen Q and anti-MuSK autoantibody competitively suppress agrin/LRP4/MuSK signaling.
Journal: Scientific reports
Published: June 22, 2015
Collagen Q is a key player for developing rational therapy for congenital myasthenia and for dissecting the mechanisms of anti-MuSK myasthenia gravis.
Journal: Journal of molecular neuroscience : MN
Published: October 10, 2013
Specific binding of collagen Q to the neuromuscular junction is exploited to cure congenital myasthenia and to explore bases of myasthenia gravis.
Journal: Chemico-biological interactions
Published: June 30, 2012
Anti-MuSK autoantibodies block binding of collagen Q to MuSK.
Journal: Neurology
Published: October 21, 2011
Showing 1-10 of 10
Last Updated: 09/26/2026