Yuanting Cui

Key Laboratory Of Geriatric Cardiovascular Cerebrovascular Disease Research
Chongqing, China

Last Updated: 09/26/2026

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Found 19 publications

Targeted metabolomic profiling and machine learning-based prediction models for persistent atrial fibrillation: a multi-center observational study.

Targeted metabolomic profiling and machine learning-based prediction models for persistent atrial fibrillation: a multi-center observational study.

Journal: PeerJ

Published: October 29, 2025

Dietary timing enhances exercise by modulating fat-muscle crosstalk via adipocyte AMPKα2 signaling.

Dietary timing enhances exercise by modulating fat-muscle crosstalk via adipocyte AMPKα2 signaling.

Journal: Cell metabolism

Published: July 16, 2024

TRPC5 deletion in the central amygdala antagonizes high-fat diet-induced obesity by increasing sympathetic innervation.

TRPC5 deletion in the central amygdala antagonizes high-fat diet-induced obesity by increasing sympathetic innervation.

Journal: International journal of obesity (2005)

Published: November 25, 2021

Recurrent moderate hypoglycemia accelerates the progression of Alzheimer's disease through impairment of the TRPC6/GLUT3 pathway.

Recurrent moderate hypoglycemia accelerates the progression of Alzheimer's disease through impairment of the TRPC6/GLUT3 pathway.

Journal: JCI insight

Published: August 30, 2021

Low-glucose-sensitive TRPC6 dysfunction drives hypoglycemia-induced cognitive impairment in diabetes.

Low-glucose-sensitive TRPC6 dysfunction drives hypoglycemia-induced cognitive impairment in diabetes.

Journal: Clinical and translational medicine

Published: July 01, 2020

Activation of the bitter taste sensor TRPM5 prevents high salt-induced cardiovascular dysfunction.

Activation of the bitter taste sensor TRPM5 prevents high salt-induced cardiovascular dysfunction.

Journal: Science China. Life sciences

Published: December 10, 2019

TRPC3 deficiency attenuates high salt-induced cardiac hypertrophy by alleviating cardiac mitochondrial dysfunction.

TRPC3 deficiency attenuates high salt-induced cardiac hypertrophy by alleviating cardiac mitochondrial dysfunction.

Journal: Biochemical and biophysical research communications

Published: August 21, 2019

High-salt intake increases TRPC3 expression and enhances TRPC3-mediated calcium influx and systolic blood pressure in hypertensive patients.

High-salt intake increases TRPC3 expression and enhances TRPC3-mediated calcium influx and systolic blood pressure in hypertensive patients.

Journal: Hypertension research : official journal of the Japanese Society of Hypertension

Published: August 19, 2019

Impairment of Bitter Taste Sensor Transient Receptor Potential Channel M5-Mediated Aversion Aggravates High-Salt Intake and Hypertension.

Impairment of Bitter Taste Sensor Transient Receptor Potential Channel M5-Mediated Aversion Aggravates High-Salt Intake and Hypertension.

Journal: Hypertension (Dallas, Tex. : 1979)

Published: August 13, 2019

Inhibition of Mitochondrial Calcium Overload by SIRT3 Prevents Obesity- or Age-Related Whitening of Brown Adipose Tissue.

Inhibition of Mitochondrial Calcium Overload by SIRT3 Prevents Obesity- or Age-Related Whitening of Brown Adipose Tissue.

Journal: Diabetes

Published: May 30, 2019

Deficiency of PKD2L1 (TRPP3) Exacerbates Pathological Cardiac Hypertrophy by Augmenting NCX1-Mediated Mitochondrial Calcium Overload.

Deficiency of PKD2L1 (TRPP3) Exacerbates Pathological Cardiac Hypertrophy by Augmenting NCX1-Mediated Mitochondrial Calcium Overload.

Journal: Cell reports

Published: October 18, 2017

Activation of Transient Receptor Potential Melastatin Subtype 8 Attenuates Cold-Induced Hypertension Through Ameliorating Vascular Mitochondrial Dysfunction.

Activation of Transient Receptor Potential Melastatin Subtype 8 Attenuates Cold-Induced Hypertension Through Ameliorating Vascular Mitochondrial Dysfunction.

Journal: Journal of the American Heart Association

Published: August 04, 2017

Showing 1-12 of 19

Last Updated: 09/26/2026