Yuji Yamanashi

Tokyo, Japan

Last Updated: 09/26/2026

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Found 34 publications

CD200R1-CD200 checkpoint inhibits phagocytosis differently from SIRPα-CD47 to suppress tumor growth.

CD200R1-CD200 checkpoint inhibits phagocytosis differently from SIRPα-CD47 to suppress tumor growth.

Journal: Nature communications
Published: October 17, 2024

DOK1 and DOK2 regulate CD8 T cell signaling and memory formation without affecting tumor cell killing.

DOK1 and DOK2 regulate CD8 T cell signaling and memory formation without affecting tumor cell killing.

Journal: Scientific reports
Published: February 21, 2024

Molecular Mechanisms Underlying the Formation and Maintenance of Neuromuscular Junctions and Development of NMJ-targeted Therapeutics

Molecular Mechanisms Underlying the Formation and Maintenance of Neuromuscular Junctions and Development of NMJ-targeted Therapeutics

Journal: Brain and nerve = Shinkei kenkyu no shinpo
Published: January 08, 2024

Characterization of disease-specific alterations in metabolites and effects of mesenchymal stromal cells on dystrophic muscles.

Characterization of disease-specific alterations in metabolites and effects of mesenchymal stromal cells on dystrophic muscles.

Journal: Frontiers in cell and developmental biology
Published: December 30, 2023

Calcium-binding protein 7 expressed in muscle negatively regulates age-related degeneration of neuromuscular junctions in mice.

Calcium-binding protein 7 expressed in muscle negatively regulates age-related degeneration of neuromuscular junctions in mice.

Journal: iScience
Published: August 30, 2023

Loss of Dok-3 in Non-tumor Cells Induces Malignant Transformation of Benign Epithelial Tumor Cells of the Intestine.

Loss of Dok-3 in Non-tumor Cells Induces Malignant Transformation of Benign Epithelial Tumor Cells of the Intestine.

Journal: Cancer research communications
Published: September 01, 2022

Adeno-associated virus-mediated expression of an inactive CaMKIIβ mutant enhances muscle mass and strength in mice.

Adeno-associated virus-mediated expression of an inactive CaMKIIβ mutant enhances muscle mass and strength in mice.

Journal: Biochemical and biophysical research communications
Published: October 23, 2021

LONRF2 is a protein quality control ubiquitin ligase whose deficiency causes late-onset neurological deficits.

LONRF2 is a protein quality control ubiquitin ligase whose deficiency causes late-onset neurological deficits.

Journal: Nature aging
Published: October 08, 2021

Effect of salbutamol on neuromuscular junction function and structure in a mouse model of DOK7 congenital myasthenia.

Effect of salbutamol on neuromuscular junction function and structure in a mouse model of DOK7 congenital myasthenia.

Journal: Human molecular genetics
Published: April 19, 2020

DOK7 Gene Therapy Enhances Neuromuscular Junction Innervation and Motor Function in Aged Mice.

DOK7 Gene Therapy Enhances Neuromuscular Junction Innervation and Motor Function in Aged Mice.

Journal: iScience
Published: April 01, 2020

Overexpression of Dok-7 in skeletal muscle enhances neuromuscular transmission with structural alterations of neuromuscular junctions: Implications in robustness of neuromuscular transmission.

Overexpression of Dok-7 in skeletal muscle enhances neuromuscular transmission with structural alterations of neuromuscular junctions: Implications in robustness of neuromuscular transmission.

Journal: Biochemical and biophysical research communications
Published: November 19, 2019

Loss of C/EBPδ enhances apoptosis of intestinal epithelial cells and exacerbates experimental colitis in mice.

Loss of C/EBPδ enhances apoptosis of intestinal epithelial cells and exacerbates experimental colitis in mice.

Journal: Genes to cells : devoted to molecular & cellular mechanisms
Published: April 14, 2019
Showing 1-12 of 34

Last Updated: 09/26/2026