Yuka Takezawa
Matsumoto, Japan
Last Updated: 09/26/2026
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Found 20 publications
Clinical impact of lilac-colored aggregates on may-Grünwald-Giemsa-stained bone marrow smears in patients with amyloid light-chain amyloidosis.
Journal: Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis
Published: March 09, 2026
Paediatric acute lymphoblastic leukaemia-associated haemophagocytic lymphohistiocytosis develops during prednisolone prephase.
Journal: British journal of haematology
Published: May 15, 2024
Clinical utility of urinary mulberry bodies/cells testing in the diagnosis of Fabry disease.
Journal: Molecular genetics and metabolism reports
Published: May 24, 2023
Immature Platelet Fraction and Its Kinetics in Neonates.
Journal: Journal of pediatric hematology/oncology
Published: November 08, 2021
Acquired dysfibrinogenemia: monoclonal λ-type IgA binding to fibrinogen caused lower functional plasma fibrinogen level and abnormal clot formation.
Journal: International journal of hematology
Published: December 05, 2019
The fibrous form of intracellular inclusion bodies in recombinant variant fibrinogen-producing cells is specific to the hepatic fibrinogen storage disease-inducible variant fibrinogen.
Journal: International journal of hematology
Published: December 08, 2016
Cell size variations of large granular lymphocyte leukemia: Implication of a small cell subtype of granular lymphocyte leukemia with STAT3 mutations.
Journal: Leukemia research
Published: December 28, 2015
Differences in the function and secretion of congenital aberrant fibrinogenemia between heterozygous γD320G (Okayama II) and γΔN319-ΔD320 (Otsu I).
Journal: Thrombosis research
Published: July 15, 2015
Novel heterozygous dysfibrinogenemia, Sumida (AαC472S), showed markedly impaired lateral aggregation of protofibrils and mildly lower functional fibrinogen levels.
Journal: Thrombosis research
Published: November 12, 2014
Recombinant γT305A fibrinogen indicates severely impaired fibrin polymerization due to the aberrant function of hole 'A' and calcium binding sites.
Journal: Thrombosis research
Published: March 07, 2014
Richter transformation in the brain from chronic lymphocytic leukemia.
Journal: Journal of clinical and experimental hematopathology : JCEH
Published: September 03, 2013
Last Updated: 09/26/2026