Yulai Fang

639 Long Mian Avenue
Nanjing, 211198, China

Last Updated: 09/26/2026

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Found 19 publications

Resolvin D5 Inhibits CXCL8 Expression in Colonic Epithelial Cells Through Activating GPR101 to Impede Neutrophil Recruitment and Consequently Alleviate Ulcerative Colitis.

Resolvin D5 Inhibits CXCL8 Expression in Colonic Epithelial Cells Through Activating GPR101 to Impede Neutrophil Recruitment and Consequently Alleviate Ulcerative Colitis.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: August 10, 2025

Bergenin inhibits γδT17 cell activation to alleviate ulcerative colitis by down-regulating fatty acid oxidation via targeting miR-124-3p.

Bergenin inhibits γδT17 cell activation to alleviate ulcerative colitis by down-regulating fatty acid oxidation via targeting miR-124-3p.

Journal: Phytomedicine : international journal of phytotherapy and phytopharmacology

Published: August 06, 2025

Isoimperatorin, a natural furanocoumarin, ameliorates ulcerative colitis by inducing Treg cell generation and promoting mucosal healing.

Isoimperatorin, a natural furanocoumarin, ameliorates ulcerative colitis by inducing Treg cell generation and promoting mucosal healing.

Journal: Molecular medicine (Cambridge, Mass.)

Published: January 21, 2025

The potential mechanism of co-administration of Scutellaria baicalensis Georgi and Rubia cordifolia L ameliorating ulcerative colitis: Integration of metabolomics, network pharmacology, and molecular docking.

The potential mechanism of co-administration of Scutellaria baicalensis Georgi and Rubia cordifolia L ameliorating ulcerative colitis: Integration of metabolomics, network pharmacology, and molecular docking.

Journal: Journal of pharmaceutical and biomedical analysis

Published: January 15, 2025

Integration of Multi-Omics and Network Pharmacology Analysis Reveals the Mechanism of Qingchang Huashi Jianpi Bushen Formula in Repairing the Epithelial Barrier of Ulcerative Colitis.

Integration of Multi-Omics and Network Pharmacology Analysis Reveals the Mechanism of Qingchang Huashi Jianpi Bushen Formula in Repairing the Epithelial Barrier of Ulcerative Colitis.

Journal: Journal of inflammation research

Published: December 20, 2024

Inflammation-fibrosis interplay in inflammatory bowel disease: mechanisms, progression, and therapeutic strategies.

Inflammation-fibrosis interplay in inflammatory bowel disease: mechanisms, progression, and therapeutic strategies.

Journal: Frontiers in pharmacology

Published: November 19, 2024

The role of mitochondrial fusion and fission in immune-mediated inflammatory diseases.

The role of mitochondrial fusion and fission in immune-mediated inflammatory diseases.

Journal: Cellular immunology

Published: June 17, 2024

Qin-Yu-Qing-Chang decoction reshapes colonic metabolism by activating PPAR-γ signaling to inhibit facultative anaerobes against DSS-induced colitis.

Qin-Yu-Qing-Chang decoction reshapes colonic metabolism by activating PPAR-γ signaling to inhibit facultative anaerobes against DSS-induced colitis.

Journal: Chinese medicine

Published: April 16, 2024

Integrated Network Pharmacology, Molecular Docking and Animal Experiment to Explore the Efficacy and Potential Mechanism of Baiyu Decoction Against Ulcerative Colitis by Enema.

Integrated Network Pharmacology, Molecular Docking and Animal Experiment to Explore the Efficacy and Potential Mechanism of Baiyu Decoction Against Ulcerative Colitis by Enema.

Journal: Drug design, development and therapy

Published: September 09, 2023

A network pharmacology approach and experimental validation to investigate the anticancer mechanism of Qi-Qin-Hu-Chang formula against colitis-associated colorectal cancer through induction of apoptosis via JNK/p38 MAPK signaling pathway.

A network pharmacology approach and experimental validation to investigate the anticancer mechanism of Qi-Qin-Hu-Chang formula against colitis-associated colorectal cancer through induction of apoptosis via JNK/p38 MAPK signaling pathway.

Journal: Journal of ethnopharmacology

Published: August 01, 2023

Mitochondrial fusion induced by transforming growth factor-β1 serves as a switch that governs the metabolic reprogramming during differentiation of regulatory T cells.

Mitochondrial fusion induced by transforming growth factor-β1 serves as a switch that governs the metabolic reprogramming during differentiation of regulatory T cells.

Journal: Redox biology

Published: January 31, 2023

AhR activation promotes Treg cell generation by enhancing Lkb1-mediated fatty acid oxidation via the Skp2/K63-ubiquitination pathway.

AhR activation promotes Treg cell generation by enhancing Lkb1-mediated fatty acid oxidation via the Skp2/K63-ubiquitination pathway.

Journal: Immunology

Published: October 25, 2022

Showing 1-12 of 19

Last Updated: 09/26/2026