Yulian Chen

NMPA Key Laboratory For Research And Evaluation Of Drug Metabolism, Guangdong Provincial Key Laboratory Of New Drug Screening
Guangzhou, 510515, China

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 10 publications

Oridonin restores hepatic lipid homeostasis in an LXRα-ATGL/EPT1 axis-dependent manner.

Oridonin restores hepatic lipid homeostasis in an LXRα-ATGL/EPT1 axis-dependent manner.

Journal: Journal of pharmaceutical analysis

Published: March 01, 2023

Restoration of lipid homeostasis between TG and PE by the LXRα-ATGL/EPT1 axis ameliorates hepatosteatosis.

Restoration of lipid homeostasis between TG and PE by the LXRα-ATGL/EPT1 axis ameliorates hepatosteatosis.

Journal: Cell death & disease

Published: September 18, 2022

Oridonin alleviates hyperbilirubinemia through activating LXRα-UGT1A1 axis.

Oridonin alleviates hyperbilirubinemia through activating LXRα-UGT1A1 axis.

Journal: Pharmacological research

Published: November 05, 2021

Effect of oridonin on oxylipins in the livers of mice with acute liver injury induced by D-galactosamine and lipopolysaccharide.

Effect of oridonin on oxylipins in the livers of mice with acute liver injury induced by D-galactosamine and lipopolysaccharide.

Journal: International immunopharmacology

Published: July 25, 2021

LXR-Mediated Regulation of Marine-Derived Piericidins Aggravates High-Cholesterol Diet-Induced Cholesterol Metabolism Disorder in Mice.

LXR-Mediated Regulation of Marine-Derived Piericidins Aggravates High-Cholesterol Diet-Induced Cholesterol Metabolism Disorder in Mice.

Journal: Journal of medicinal chemistry

Published: July 12, 2021

Oridonin alleviates d-GalN/LPS-induced acute liver injury by inhibiting NLRP3 inflammasome.

Oridonin alleviates d-GalN/LPS-induced acute liver injury by inhibiting NLRP3 inflammasome.

Journal: Drug development research

Published: June 17, 2020

Metabolism and Toxicity of Emodin: Genome-Wide Association Studies Reveal Hepatocyte Nuclear Factor 4α Regulates UGT2B7 and Emodin Glucuronidation.

Metabolism and Toxicity of Emodin: Genome-Wide Association Studies Reveal Hepatocyte Nuclear Factor 4α Regulates UGT2B7 and Emodin Glucuronidation.

Journal: Chemical research in toxicology

Published: June 16, 2020

Regulation of cytochrome P450 4F11 expression by liver X receptor alpha.

Regulation of cytochrome P450 4F11 expression by liver X receptor alpha.

Journal: International immunopharmacology

Published: June 07, 2020

Gender Differences in the Hepatotoxicity and Toxicokinetics of Emodin: The Potential Mechanisms Mediated by UGT2B7 and MRP2.

Gender Differences in the Hepatotoxicity and Toxicokinetics of Emodin: The Potential Mechanisms Mediated by UGT2B7 and MRP2.

Journal: Molecular pharmaceutics

Published: July 17, 2018

Emodin-induced hepatotoxicity was exacerbated by probenecid through inhibiting UGTs and MRP2.

Emodin-induced hepatotoxicity was exacerbated by probenecid through inhibiting UGTs and MRP2.

Journal: Toxicology and applied pharmacology

Published: June 19, 2018

Showing 1-10 of 10

Last Updated: 09/26/2026