Yunman Wang

164 Lanxi Road
Putuo, 200062, China

Last Updated: 09/26/2026

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Found 22 publications

Astragaloside IV attenuates podocyte apoptosis through ameliorating mitochondrial dysfunction by up-regulated Nrf2-ARE/TFAM signaling in diabetic kidney disease.

Astragaloside IV attenuates podocyte apoptosis through ameliorating mitochondrial dysfunction by up-regulated Nrf2-ARE/TFAM signaling in diabetic kidney disease.

Journal: Free radical biology & medicine

Published: January 03, 2023

Astragaloside IV attenuates high glucose-induced NF-κB-mediated inflammation through activation of PI3K/AKT-ERK-dependent Nrf2/ARE signaling pathway in glomerular mesangial cells.

Astragaloside IV attenuates high glucose-induced NF-κB-mediated inflammation through activation of PI3K/AKT-ERK-dependent Nrf2/ARE signaling pathway in glomerular mesangial cells.

Journal: Phytotherapy research : PTR

Published: November 28, 2022

Knowledge-Based Discovery of the Role and Mechanism of Resveratrol in Improving Glomerular Tether Cell Proliferation and Apoptosis in Diabetic Nephropathy.

Knowledge-Based Discovery of the Role and Mechanism of Resveratrol in Improving Glomerular Tether Cell Proliferation and Apoptosis in Diabetic Nephropathy.

Journal: Journal of healthcare engineering

Published: January 12, 2022

Serum chromogranin A correlated with albuminuria in diabetic patients and is associated with early diabetic nephropathy.

Serum chromogranin A correlated with albuminuria in diabetic patients and is associated with early diabetic nephropathy.

Journal: BMC nephrology

Published: August 04, 2021

Klotho ameliorates diabetic nephropathy by activating Nrf2 signaling pathway in podocytes.

Klotho ameliorates diabetic nephropathy by activating Nrf2 signaling pathway in podocytes.

Journal: Biochemical and biophysical research communications

Published: October 17, 2020

Astragaloside IV protects against podocyte apoptosis by inhibiting oxidative stress via activating PPARγ-Klotho-FoxO1 axis in diabetic nephropathy.

Astragaloside IV protects against podocyte apoptosis by inhibiting oxidative stress via activating PPARγ-Klotho-FoxO1 axis in diabetic nephropathy.

Journal: Life sciences

Published: August 03, 2020

Astragaloside IV inhibits palmitic acid-induced apoptosis through regulation of calcium homeostasis in mice podocytes.

Astragaloside IV inhibits palmitic acid-induced apoptosis through regulation of calcium homeostasis in mice podocytes.

Journal: Molecular biology reports

Published: June 14, 2020

Serum and urine metabolomics reveal potential biomarkers of T2DM patients with nephropathy.

Serum and urine metabolomics reveal potential biomarkers of T2DM patients with nephropathy.

Journal: Annals of translational medicine

Published: April 21, 2020

ApoL1 induces kidney inflammation through RIG-I/NF-κB activation.

ApoL1 induces kidney inflammation through RIG-I/NF-κB activation.

Journal: Biochemical and biophysical research communications

Published: March 25, 2020

Huangqi decoction inhibits hyperglycemia-induced podocyte apoptosis by down-regulated Nox4/p53/Bax signaling in vitro and in vivo.

Huangqi decoction inhibits hyperglycemia-induced podocyte apoptosis by down-regulated Nox4/p53/Bax signaling in vitro and in vivo.

Journal: American journal of translational research

Published: September 10, 2018

Huangqi (astragalus) decoction ameliorates diabetic nephropathy via IRS1-PI3K-GLUT signaling pathway.

Huangqi (astragalus) decoction ameliorates diabetic nephropathy via IRS1-PI3K-GLUT signaling pathway.

Journal: American journal of translational research

Published: April 28, 2018

Indoxyl sulfate potentiates endothelial dysfunction via reciprocal role for reactive oxygen species and RhoA/ROCK signaling in 5/6 nephrectomized rats.

Indoxyl sulfate potentiates endothelial dysfunction via reciprocal role for reactive oxygen species and RhoA/ROCK signaling in 5/6 nephrectomized rats.

Journal: Free radical research

Published: March 10, 2017

Showing 1-12 of 22

Last Updated: 09/26/2026