Yuting Fan

Tongji University School Of Medicine 301 Yanchang Road
Zhabei, 200072, China

Last Updated: 09/26/2026

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Found 8 publications

Role of Microvesicles From Bone Marrow Mesenchymal Stem Cells in Acute Pancreatitis.

Role of Microvesicles From Bone Marrow Mesenchymal Stem Cells in Acute Pancreatitis.

Journal: Pancreas

Published: October 25, 2016

Reverse-migrated neutrophils regulated by JAM-C are involved in acute pancreatitis-associated lung injury.

Reverse-migrated neutrophils regulated by JAM-C are involved in acute pancreatitis-associated lung injury.

Journal: Scientific reports

Published: June 23, 2015

Role of bone marrow mesenchymal stem cells in L-arg-induced acute pancreatitis: effects and possible mechanisms.

Role of bone marrow mesenchymal stem cells in L-arg-induced acute pancreatitis: effects and possible mechanisms.

Journal: International journal of clinical and experimental pathology

Published: March 05, 2015

Retinoic Acid Ameliorates Pancreatic Fibrosis and Inhibits the Activation of Pancreatic Stellate Cells in Mice with Experimental Chronic Pancreatitis via Suppressing the Wnt/β-Catenin Signaling Pathway.

Retinoic Acid Ameliorates Pancreatic Fibrosis and Inhibits the Activation of Pancreatic Stellate Cells in Mice with Experimental Chronic Pancreatitis via Suppressing the Wnt/β-Catenin Signaling Pathway.

Journal: PloS one

Published: January 15, 2015

Astragaloside IV ameliorates acute pancreatitis in rats by inhibiting the activation of nuclear factor-κB.

Astragaloside IV ameliorates acute pancreatitis in rats by inhibiting the activation of nuclear factor-κB.

Journal: International journal of molecular medicine

Published: September 06, 2014

Diosmetin ameliorates the severity of cerulein-induced acute pancreatitis in mice by inhibiting the activation of the nuclear factor-κB.

Diosmetin ameliorates the severity of cerulein-induced acute pancreatitis in mice by inhibiting the activation of the nuclear factor-κB.

Journal: International journal of clinical and experimental pathology

Published: February 01, 2014

Pancreatic acinar cells-derived cyclophilin A promotes pancreatic damage by activating NF-κB pathway in experimental pancreatitis.

Pancreatic acinar cells-derived cyclophilin A promotes pancreatic damage by activating NF-κB pathway in experimental pancreatitis.

Journal: Biochemical and biophysical research communications

Published: December 24, 2013

Imbalance of Wnt/Dkk negative feedback promotes persistent activation of pancreatic stellate cells in chronic pancreatitis.

Imbalance of Wnt/Dkk negative feedback promotes persistent activation of pancreatic stellate cells in chronic pancreatitis.

Journal: PloS one

Published: October 08, 2013

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Last Updated: 09/26/2026