Yuya Araki
Yushima, 1138519, Japan
Last Updated: 09/26/2026
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Found 9 publications
Decreased KLHL3 expression is involved in the pathogenesis of pseudohypoaldosteronism type II caused by cullin 3 mutation in vivo.
Journal: Clinical and experimental nephrology
Published: April 07, 2018
Drug-Repositioning Screening for Keap1-Nrf2 Binding Inhibitors using Fluorescence Correlation Spectroscopy.
Journal: Scientific reports
Published: October 10, 2016
KLHL3 Knockout Mice Reveal the Physiological Role of KLHL3 and the Pathophysiology of Pseudohypoaldosteronism Type II Caused by Mutant KLHL3.
Journal: Molecular and cellular biology
Published: September 25, 2016
Impaired degradation of WNK by Akt and PKA phosphorylation of KLHL3.
Journal: Biochemical and biophysical research communications
Published: September 10, 2015
Kelch-Like Protein 2 Mediates Angiotensin II-With No Lysine 3 Signaling in the Regulation of Vascular Tonus.
Journal: Journal of the American Society of Nephrology : JASN
Published: July 02, 2014
WNK4 is the major WNK positively regulating NCC in the mouse kidney.
Journal: Bioscience reports
Published: March 25, 2014
Decrease of WNK4 ubiquitination by disease-causing mutations of KLHL3 through different molecular mechanisms.
Journal: Biochemical and biophysical research communications
Published: July 29, 2013
Impaired KLHL3-mediated ubiquitination of WNK4 causes human hypertension.
Journal: Cell reports
Published: November 28, 2012
Showing 1-9 of 9
Last Updated: 09/26/2026