Zhaojing Lu

Key Laboratory Of Molecular Biophysics Of Ministry Of Education
Wuhan, 430074, China

Last Updated: 09/26/2026

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Found 9 publications

Reply to Corbeil et al.: Deletion of the transmembrane protein Prom1b in zebrafish disrupts outer-segment morphogenesis and causes photoreceptor degeneration.

Reply to Corbeil et al.: Deletion of the transmembrane protein Prom1b in zebrafish disrupts outer-segment morphogenesis and causes photoreceptor degeneration.

Journal: The Journal of biological chemistry

Published: November 10, 2019

Deletion of the transmembrane protein Prom1b in zebrafish disrupts outer-segment morphogenesis and causes photoreceptor degeneration.

Deletion of the transmembrane protein Prom1b in zebrafish disrupts outer-segment morphogenesis and causes photoreceptor degeneration.

Journal: The Journal of biological chemistry

Published: March 28, 2019

BCAS2 is essential for hematopoietic stem and progenitor cell maintenance during zebrafish embryogenesis.

BCAS2 is essential for hematopoietic stem and progenitor cell maintenance during zebrafish embryogenesis.

Journal: Blood

Published: September 25, 2018

CERKL regulates autophagy via the NAD-dependent deacetylase SIRT1.

CERKL regulates autophagy via the NAD-dependent deacetylase SIRT1.

Journal: Autophagy

Published: September 13, 2018

Knockout of ush2a gene in zebrafish causes hearing impairment and late onset rod-cone dystrophy.

Knockout of ush2a gene in zebrafish causes hearing impairment and late onset rod-cone dystrophy.

Journal: Human genetics

Published: March 22, 2018

HSF4 regulates lens fiber cell differentiation by activating p53 and its downstream regulators.

HSF4 regulates lens fiber cell differentiation by activating p53 and its downstream regulators.

Journal: Cell death & disease

Published: April 25, 2017

CERKL gene knockout disturbs photoreceptor outer segment phagocytosis and causes rod-cone dystrophy in zebrafish.

CERKL gene knockout disturbs photoreceptor outer segment phagocytosis and causes rod-cone dystrophy in zebrafish.

Journal: Human molecular genetics

Published: January 22, 2017

Ablation of EYS in zebrafish causes mislocalisation of outer segment proteins, F-actin disruption and cone-rod dystrophy.

Ablation of EYS in zebrafish causes mislocalisation of outer segment proteins, F-actin disruption and cone-rod dystrophy.

Journal: Scientific reports

Published: November 02, 2016

Pathogenic mutations in retinitis pigmentosa 2 predominantly result in loss of RP2 protein stability in humans and zebrafish.

Pathogenic mutations in retinitis pigmentosa 2 predominantly result in loss of RP2 protein stability in humans and zebrafish.

Journal: The Journal of biological chemistry

Published: September 25, 2016

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Last Updated: 09/26/2026