Zhenheng Wang

Nanjing, China

Last Updated: 09/26/2026

Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 14 publications

Correction to"Specifically Formed Corona on Silica Nanoparticles Enhances Transforming Growth Factor beta 1 Activity in Triggering Lung Fibrosis".

Correction to"Specifically Formed Corona on Silica Nanoparticles Enhances Transforming Growth Factor beta 1 Activity in Triggering Lung Fibrosis".

Journal: ACS nano
Published: October 11, 2024

STAT3/IL-6 dependent induction of inflammatory response in osteoblast and osteoclast formation in nanoscale wear particle-induced aseptic prosthesis loosening.

STAT3/IL-6 dependent induction of inflammatory response in osteoblast and osteoclast formation in nanoscale wear particle-induced aseptic prosthesis loosening.

Journal: Biomaterials science
Published: December 18, 2020

Probiotics protect mice from CoCrMo particles-induced osteolysis.

Probiotics protect mice from CoCrMo particles-induced osteolysis.

Journal: International journal of nanomedicine
Published: August 11, 2017

Enhanced Uptake of Fe₃O₄ Nanoparticles by Intestinal Epithelial Cells in a State of Inflammation.

Enhanced Uptake of Fe₃O₄ Nanoparticles by Intestinal Epithelial Cells in a State of Inflammation.

Journal: Molecules (Basel, Switzerland)
Published: June 05, 2017

The metal nanoparticle-induced inflammatory response is regulated by SIRT1 through NF-κB deacetylation in aseptic loosening.

The metal nanoparticle-induced inflammatory response is regulated by SIRT1 through NF-κB deacetylation in aseptic loosening.

Journal: International journal of nanomedicine
Published: May 30, 2017

Specifically Formed Corona on Silica Nanoparticles Enhances Transforming Growth Factor β1 Activity in Triggering Lung Fibrosis.

Specifically Formed Corona on Silica Nanoparticles Enhances Transforming Growth Factor β1 Activity in Triggering Lung Fibrosis.

Journal: ACS nano
Published: January 14, 2017

SIRT1 protects osteoblasts against particle-induced inflammatory responses and apoptosis in aseptic prosthesis loosening.

SIRT1 protects osteoblasts against particle-induced inflammatory responses and apoptosis in aseptic prosthesis loosening.

Journal: Acta biomaterialia
Published: July 27, 2016

TiAl6V4 particles promote osteoclast formation via autophagy-mediated downregulation of interferon-beta in osteocytes.

TiAl6V4 particles promote osteoclast formation via autophagy-mediated downregulation of interferon-beta in osteocytes.

Journal: Acta biomaterialia
Published: July 13, 2016

Autophagy mediated TiAl₆V₄ particle-induced peri-implant osteolysis by promoting expression of TNF-α.

Autophagy mediated TiAl₆V₄ particle-induced peri-implant osteolysis by promoting expression of TNF-α.

Journal: Biochemical and biophysical research communications
Published: March 07, 2016

Autophagy mediated CoCrMo particle-induced peri-implant osteolysis by promoting osteoblast apoptosis.

Autophagy mediated CoCrMo particle-induced peri-implant osteolysis by promoting osteoblast apoptosis.

Journal: Autophagy
Published: November 14, 2015

Expression of XBP1s in fibroblasts is critical for TiAl6 V4 particle-induced RANKL expression and osteolysis.

Expression of XBP1s in fibroblasts is critical for TiAl6 V4 particle-induced RANKL expression and osteolysis.

Journal: Journal of orthopaedic research : official publication of the Orthopaedic Research Society
Published: June 15, 2015

ER Stress Mediates TiAl6V4 Particle-Induced Peri-Implant Osteolysis by Promoting RANKL Expression in Fibroblasts.

ER Stress Mediates TiAl6V4 Particle-Induced Peri-Implant Osteolysis by Promoting RANKL Expression in Fibroblasts.

Journal: PloS one
Published: April 17, 2015
Showing 1-12 of 14

Last Updated: 09/26/2026