Zhenqiong Guan

Beijing, China

Last Updated: 09/26/2026

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Found 10 publications

A viral necrosome mediates direct RIPK3 activation to promote inflammatory necroptosis.

A viral necrosome mediates direct RIPK3 activation to promote inflammatory necroptosis.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: May 27, 2025

Correction: RVFV virulence factor NSs triggers the mitochondrial MCL-1-BAK axis to activate pathogenic NLRP3 pyroptosis.

Correction: RVFV virulence factor NSs triggers the mitochondrial MCL-1-BAK axis to activate pathogenic NLRP3 pyroptosis.

Journal: PLoS pathogens

Published: April 24, 2025

Rift Valley fever virus coordinates the assembly of a programmable E3 ligase to promote viral replication.

Rift Valley fever virus coordinates the assembly of a programmable E3 ligase to promote viral replication.

Journal: Cell

Published: February 23, 2025

Rift Valley fever virus coordinates the assembly of a programmable E3 ligase to promote viral replication.

Rift Valley fever virus coordinates the assembly of a programmable E3 ligase to promote viral replication.

Journal: Cell

Published: March 18, 2024

RVFV virulence factor NSs triggers the mitochondrial MCL-1-BAK axis to activate pathogenic NLRP3 pyroptosis.

RVFV virulence factor NSs triggers the mitochondrial MCL-1-BAK axis to activate pathogenic NLRP3 pyroptosis.

Journal: PLoS pathogens

Published: February 04, 2024

Rift Valley Fever Virus Nucleoprotein Triggers Autophagy to Dampen Antiviral Innate Immune Responses.

Rift Valley Fever Virus Nucleoprotein Triggers Autophagy to Dampen Antiviral Innate Immune Responses.

Journal: Journal of virology

Published: March 20, 2023

SARS-CoV-2 Z-RNA activates the ZBP1-RIPK3 pathway to promote virus-induced inflammatory responses.

SARS-CoV-2 Z-RNA activates the ZBP1-RIPK3 pathway to promote virus-induced inflammatory responses.

Journal: Cell research

Published: June 20, 2022

SARS-CoV-2 triggers inflammatory responses and cell death through caspase-8 activation.

SARS-CoV-2 triggers inflammatory responses and cell death through caspase-8 activation.

Journal: Signal transduction and targeted therapy

Published: July 26, 2020

NSs Filament Formation Is Important but Not Sufficient for RVFV Virulence In Vivo.

NSs Filament Formation Is Important but Not Sufficient for RVFV Virulence In Vivo.

Journal: Viruses

Published: August 05, 2019

Host factor heat-shock protein 90 contributes to baculovirus budded virus morphogenesis via facilitating nuclear actin polymerization.

Host factor heat-shock protein 90 contributes to baculovirus budded virus morphogenesis via facilitating nuclear actin polymerization.

Journal: Virology

Published: February 25, 2019

Showing 1-10 of 10

Last Updated: 09/26/2026