Zhimei M. Qiu
Zunyi, 563000, China
Last Updated: 09/26/2026
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Found 20 publications
Nomogram incorporating OCT-based plaque morphology and computational physiology features for predicting major adverse cardiovascular events in patients with acute coronary syndrome
Journal: Zhonghua xin xue guan bing za zhi
Published: May 20, 2026
Lactylation formation, gene regulation, biological functions, and clinical relevance.
Journal: Histology and histopathology
Published: September 05, 2025
Harnessing mRNA for heart health: a new era in cardiovascular treatment.
Journal: Theranostics
Published: February 04, 2025
The ubiquitin system in heart and vessel pathobiology: From mechanisms to therapeutic strategies.
Journal: Genes & diseases
Published: December 04, 2024
YTHDC1 Mitigates Apoptosis in Bone Marrow Mesenchymal Stem Cells by Inhibiting NfƙBiα and Augmenting Cardiac Function Following Myocardial Infarction.
Journal: Cell transplantation
Published: October 28, 2024
The role of cellular senescence in cardiovascular disease.
Journal: Cell death discovery
Published: September 28, 2024
Nanomedicine in cardiology: Precision drug delivery for enhanced patient outcomes.
Journal: Life sciences
Published: June 24, 2024
Non-target lesion progression: Unveiling critical predictors and outcomes in patients with in-stent restenosis.
Journal: International journal of cardiology
Published: April 24, 2024
Loss of m6A demethylase ALKBH5 alleviates hypoxia-induced pulmonary arterial hypertension via inhibiting Cyp1a1 mRNA decay.
Journal: Journal of molecular and cellular cardiology
Published: September 20, 2023
Naturally Occurring Atherosclerosis Progression and In-stent Restenosis: Exploring Histomorphologic Associations Using Optical Coherence Tomography.
Journal: Journal of cardiovascular pharmacology
Published: July 14, 2023
Semaphorin3A Exacerbates Cardiac Microvascular Rarefaction in Pressure Overload-Induced Heart Disease.
Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: November 20, 2022
Methyltransferase like 3-mediated N6-methylatidin methylation inhibits vascular smooth muscle cells phenotype switching via promoting phosphatidylinositol 3-kinase mRNA decay.
Journal: Frontiers in cardiovascular medicine
Published: April 05, 2022
Last Updated: 09/26/2026