Zhiyao Zhao

Guangzhou, China

Last Updated: 09/26/2026

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Found 17 publications

Author Correction: Genetic variants in UNC93B1 predispose to childhood-onset systemic lupus erythematosus.

Author Correction: Genetic variants in UNC93B1 predispose to childhood-onset systemic lupus erythematosus.

Journal: Nature immunology

Published: September 09, 2024

Targeting the TRIM14/USP14 Axis Enhances Immunotherapy Efficacy by Inducing Autophagic Degradation of PD-L1.

Targeting the TRIM14/USP14 Axis Enhances Immunotherapy Efficacy by Inducing Autophagic Degradation of PD-L1.

Journal: Cancer research

Published: December 13, 2023

Genetic variants in UNC93B1 predispose to childhood-onset systemic lupus erythematosus.

Genetic variants in UNC93B1 predispose to childhood-onset systemic lupus erythematosus.

Journal: Nature immunology

Published: September 12, 2023

P21-activated kinase 1 (PAK1)-mediated cytoskeleton rearrangement promotes SARS-CoV-2 entry and ACE2 autophagic degradation.

P21-activated kinase 1 (PAK1)-mediated cytoskeleton rearrangement promotes SARS-CoV-2 entry and ACE2 autophagic degradation.

Journal: Signal transduction and targeted therapy

Published: February 01, 2023

Aging-associated accumulation of mitochondrial DNA mutations in tumor origin.

Aging-associated accumulation of mitochondrial DNA mutations in tumor origin.

Journal: Life medicine

Published: April 29, 2022

Metabolic enzyme UAP1 mediates IRF3 pyrophosphorylation to facilitate innate immune response.

Metabolic enzyme UAP1 mediates IRF3 pyrophosphorylation to facilitate innate immune response.

Journal: Molecular cell

Published: April 06, 2022

TRIM14 inhibits OPTN-mediated autophagic degradation of KDM4D to epigenetically regulate inflammation.

TRIM14 inhibits OPTN-mediated autophagic degradation of KDM4D to epigenetically regulate inflammation.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: January 05, 2022

Suppression of ACE2 SUMOylation protects against SARS-CoV-2 infection through TOLLIP-mediated selective autophagy.

Suppression of ACE2 SUMOylation protects against SARS-CoV-2 infection through TOLLIP-mediated selective autophagy.

Journal: Nature communications

Published: November 11, 2021

RNA-induced liquid phase separation of SARS-CoV-2 nucleocapsid protein facilitates NF-κB hyper-activation and inflammation.

RNA-induced liquid phase separation of SARS-CoV-2 nucleocapsid protein facilitates NF-κB hyper-activation and inflammation.

Journal: Signal transduction and targeted therapy

Published: January 10, 2021

Main protease of SARS-CoV-2 serves as a bifunctional molecule in restricting type I interferon antiviral signaling.

Main protease of SARS-CoV-2 serves as a bifunctional molecule in restricting type I interferon antiviral signaling.

Journal: Signal transduction and targeted therapy

Published: July 17, 2020

Selective autophagy controls the stability of transcription factor IRF3 to balance type I interferon production and immune suppression.

Selective autophagy controls the stability of transcription factor IRF3 to balance type I interferon production and immune suppression.

Journal: Autophagy

Published: June 02, 2020

Excessive deubiquitination of NLRP3-R779C variant contributes to very-early-onset inflammatory bowel disease development.

Excessive deubiquitination of NLRP3-R779C variant contributes to very-early-onset inflammatory bowel disease development.

Journal: The Journal of allergy and clinical immunology

Published: February 28, 2020

Showing 1-12 of 17

Last Updated: 09/26/2026