Zhong-shi Lyu

Beijing Key Laboratory Of Cell And Gene Therapy For Hematologic Malignancies
Beijing, China

Last Updated: 09/26/2026

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Found 16 publications

Bone marrow SELENOP+ macrophages support hematopoiesis after transplantation via GAS6-AXL signaling pathway.

Bone marrow SELENOP+ macrophages support hematopoiesis after transplantation via GAS6-AXL signaling pathway.

Journal: Cancer letters

Published: January 21, 2026

TGF-β1-triggered maladaptive bone marrow endothelium impedes hematopoietic recovery.

TGF-β1-triggered maladaptive bone marrow endothelium impedes hematopoietic recovery.

Journal: Signal transduction and targeted therapy

Published: April 06, 2025

The Impact of Blast Percentage on Posttransplant Outcomes of Patients With Myelodysplastic Syndromes: A Retrospective Analysis Within the IPSS-M Framework.

The Impact of Blast Percentage on Posttransplant Outcomes of Patients With Myelodysplastic Syndromes: A Retrospective Analysis Within the IPSS-M Framework.

Journal: Clinical transplantation

Published: February 27, 2025

Correction: M2 macrophages, but not M1 macrophages, support megakaryopoiesis by upregulating PI3K-AKT pathway activity.

Correction: M2 macrophages, but not M1 macrophages, support megakaryopoiesis by upregulating PI3K-AKT pathway activity.

Journal: Signal transduction and targeted therapy

Published: September 12, 2024

Inhibition of TGF-β signaling in bone marrow endothelial cells promotes hematopoietic recovery in acute myeloid leukemia patients.

Inhibition of TGF-β signaling in bone marrow endothelial cells promotes hematopoietic recovery in acute myeloid leukemia patients.

Journal: Cancer letters

Published: September 12, 2024

TGF-β inhibition restores hematopoiesis and immune balance via bone marrow EPCs in aplastic anemia.

TGF-β inhibition restores hematopoiesis and immune balance via bone marrow EPCs in aplastic anemia.

Journal: Experimental & molecular medicine

Published: August 19, 2024

Activation of PPARδ in bone marrow endothelial progenitor cells improves their hematopoiesis-supporting ability after myelosuppressive injury.

Activation of PPARδ in bone marrow endothelial progenitor cells improves their hematopoiesis-supporting ability after myelosuppressive injury.

Journal: Cancer letters

Published: March 07, 2024

Bone Marrow Endothelial Progenitor Cells remodelling facilitates normal hematopoiesis during Acute Myeloid Leukemia Complete Remission.

Bone Marrow Endothelial Progenitor Cells remodelling facilitates normal hematopoiesis during Acute Myeloid Leukemia Complete Remission.

Journal: Nature communications

Published: February 12, 2024

Repair of dysfunctional bone marrow endothelial cells alleviates aplastic anemia.

Repair of dysfunctional bone marrow endothelial cells alleviates aplastic anemia.

Journal: Science China. Life sciences

Published: October 16, 2022

Dysfunctional bone marrow endothelial progenitor cells are involved in patients with myelodysplastic syndromes.

Dysfunctional bone marrow endothelial progenitor cells are involved in patients with myelodysplastic syndromes.

Journal: Journal of translational medicine

Published: February 03, 2022

The glycolytic enzyme PFKFB3 determines bone marrow endothelial progenitor cell damage after chemotherapy and irradiation.

The glycolytic enzyme PFKFB3 determines bone marrow endothelial progenitor cell damage after chemotherapy and irradiation.

Journal: Haematologica

Published: August 02, 2021

M2 macrophages, but not M1 macrophages, support megakaryopoiesis by upregulating PI3K-AKT pathway activity.

M2 macrophages, but not M1 macrophages, support megakaryopoiesis by upregulating PI3K-AKT pathway activity.

Journal: Signal transduction and targeted therapy

Published: October 22, 2020

Showing 1-12 of 16

Last Updated: 09/26/2026