Zixiang Wang
Jinan, China
Last Updated: 09/26/2026
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Found 18 publications
Splice-switching of the oncogenic BCS1L isoform suppresses ovarian cancer progression by disrupting mitochondrial function.
Journal: Cell death & disease
Published: October 10, 2025
TMC7 deficiency causes acrosome biogenesis defects and male infertility in mice.
Journal: eLife
Published: September 13, 2024
Author Correction: Therapy-induced secretion of spliceosomal components mediates pro-survival crosstalk between ovarian cancer cells.
Journal: Nature communications
Published: July 31, 2024
Targeting tumor-associated macrophage-derived CD74 improves efficacy of neoadjuvant chemotherapy in combination with PD-1 blockade for cervical cancer.
Journal: Journal for immunotherapy of cancer
Published: July 07, 2024
Splicing Factor PQBP1 Curtails BAX Expression to Promote Ovarian Cancer Progression.
Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: August 31, 2023
Spliceosome component Usp39 contributes to hepatic lipid homeostasis through the regulation of autophagy.
Journal: Nature communications
Published: February 14, 2023
The splicing factor SF3B4 drives proliferation and invasion in cervical cancer by regulating SPAG5.
Journal: Cell death discovery
Published: February 11, 2022
Therapy-induced secretion of spliceosomal components mediates pro-survival crosstalk between ovarian cancer cells.
Journal: Nature communications
Published: February 04, 2022
Bud31-mediated alternative splicing is required for spermatogonial stem cell self-renewal and differentiation.
Journal: Cell death and differentiation
Published: February 04, 2022
MEX3A promotes the malignant progression of ovarian cancer by regulating intron retention in TIMELESS.
Journal: Cell death & disease
Published: December 27, 2021
Last Updated: 09/26/2026