Zyta Majkutewicz-Banecka
Gdansk, PM, Poland
Last Updated: 09/26/2026
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Found 10 publications
Evidence for interactions between homocysteine and genistein: insights into stroke risk and potential treatment.
Journal: Metabolic brain disease
Published: January 03, 2017
Glycosaminoglycans and mucopolysaccharidosis type III.
Journal: Frontiers in bioscience (Landmark edition)
Published: April 22, 2016
The model homologue of the partially defective human 5,10-methylenetetrahydrofolate reductase, considered as a risk factor for stroke due to increased homocysteine level, can be protected and reactivated by heat shock proteins.
Journal: Metabolic brain disease
Published: January 13, 2016
Genistein inhibits activities of methylenetetrahydrofolate reductase and lactate dehydrogenase, enzymes which use NADH as a substrate.
Journal: Biochemical and biophysical research communications
Published: July 30, 2015
Increased levels of antibodies against heat shock proteins in stroke patients.
Journal: Acta biochimica Polonica
Published: May 07, 2014
Factors and processes modulating phenotypes in neuronopathic lysosomal storage diseases.
Journal: Metabolic brain disease
Published: August 16, 2013
Homocysteine, heat shock proteins, genistein and vitamins in ischemic stroke--pathogenic and therapeutic implications.
Journal: Acta biochimica Polonica
Published: November 19, 2012
Comparison of siRNA-mediated silencing of glycosaminoglycan synthesis genes and enzyme replacement therapy for mucopolysaccharidosis in cell culture studies.
Journal: Acta biochimica Polonica
Published: November 18, 2012
Role of heat-shock proteins and cobalamine in maintaining methionine synthase activity.
Journal: Acta biochimica Polonica
Published: November 15, 2012
Putative biological mechanisms of efficiency of substrate reduction therapies for mucopolysaccharidoses.
Journal: Archivum immunologiae et therapiae experimentalis
Published: February 28, 2012
Showing 1-10 of 10
Last Updated: 09/26/2026