Ectropion is the turning out of the eyelid so that the inner surface is exposed. It most often affects the lower eyelid.
Ectropion is very often caused by the aging process. The connective (supporting) tissue of the eyelid becomes weak. This causes the lid to turn out so that the inside of the lower lid is no longer against the eyeball. It can also be caused by:
If you have ectropion, you will most likely have excess tearing. This happens because the eye gets dry, then makes more tears. The excess tears can't get into the tear drainage duct. Therefore, they build up inside the lower lid and then spill over the edge of the lid onto the cheek.
Artificial tears (a lubricant) may ease dryness and keep the cornea moist. Ointment may be helpful when the eye can't close all of the way, such as when you are asleep. Surgery is very often effective. When ectropion is related to aging or paralysis, the surgeon can tighten the muscles that hold the eyelids in place. If the condition is due to scarring of the skin, a skin graft or laser treatment may be used. The surgery is most often done in the office or at an outpatient surgery center. A medicine is used to numb the area (local anesthesia) before the surgery.
The outcome is very often good with treatment.
Corneal dryness and irritation may lead to:
Corneal ulcers can cause vision loss.
Call your provider if you have symptoms of ectropion.
If you have ectropion, get emergency medical help if you have:
Most cases cannot be prevented. You may want to use artificial tears or ointments to prevent injury to the cornea, especially if you are waiting for a more permanent treatment.
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Nicoli F, Orfaniotis G, Ciudad P, et al. Correction of cicatricial ectropion using non-ablative fractional laser resurfacing. Lasers Med Sci. 2019;34(1):79-84. PMID: 30056585 pubmed.ncbi.nlm.nih.gov/30056585/.
Olitsky SE, Marsh JM. Abnormalities of the lids. In: Kliegman RM, St. Geme JW, Blum NJ, Shah SS, Tasker RC, Wilson KM, eds. Nelson Textbook of Pediatrics. 21st ed. Philadelphia, PA: Elsevier; 2020:chap 642.