An epidural hematoma (EDH) is bleeding between the inside of the skull and the outer covering of the brain (called the dura).
Extradural hematoma; Extradural hemorrhage; Epidural hemorrhage; EDH
An EDH is often caused by a skull fracture during childhood or adolescence. The membrane covering the brain is not as closely attached to the skull as it is in older people and children younger than 2 years. Therefore, this type of bleeding is more common in young people.
An EDH can also occur due to rupture of a blood vessel, usually an artery. The blood vessel then bleeds into the space between the dura and the skull.
The affected vessels are often torn by skull fractures. The fractures are most often the result of a severe head injury, such as those caused by motorcycle, bicycle, skateboard, snow boarding, or automobile accidents.
Rapid bleeding causes a collection of blood (hematoma) that presses on the brain. The pressure inside the head (intracranial pressure, ICP) increases quickly. This pressure may result in more brain injury.
Contact a health care provider for any head injury that results in even a brief loss of consciousness, or if there are any other symptoms after a head injury (even without loss of consciousness).
The typical pattern of symptoms that indicate an EDH is a loss of consciousness, followed by alertness, then loss of consciousness again. But this pattern may NOT appear in all people.
The most important symptoms of an EDH are:
The symptoms usually occur within minutes to hours after a head injury and indicate an emergency situation.
Sometimes, bleeding does not start for hours after a head injury. The symptoms of pressure on the brain also do not occur right away.
An EDH is an emergency condition. Treatment goals include:
Life support measures may be required. Emergency surgery is often necessary to reduce pressure within the brain. This may include drilling a small hole in the skull to relieve pressure and allow blood to drain outside the skull.
Large hematomas or solid blood clots may need to be removed through a larger opening in the skull (craniotomy).
Medicines used in addition to surgery will vary according to the type and severity of symptoms and brain damage that occurs.
Antiseizure medicines may be used to control or prevent seizures. Some medicines called hyperosmotic agents may be used to reduce brain swelling.
For people on blood thinners or with bleeding disorders, treatments to prevent further bleeding might be needed.
An EDH has a high risk of death without prompt surgical intervention. Even with prompt medical attention, a significant risk of death and disability remains.
There is a risk of permanent brain injury, even if EDH is treated. Symptoms (such as seizures) may persist for several months, even after treatment. In time they may become less frequent or disappear. Seizures may begin up to 2 years after the injury.
In adults, most recovery occurs in the first 6 months. Usually there is some improvement over 2 years.
If there is brain damage, full recovery isn't likely. Other complications include permanent symptoms, such as:
Go to the emergency room or call 911 or the local emergency number if symptoms of EDH occur.
Spinal injuries often occur with head injuries. If you must move the person before help arrives, try to keep his or her neck still.
Call the provider if these symptoms persist after treatment:
Go to the emergency room or call 911 or the local emergency number if these symptoms develop after treatment:
An EDH may not be preventable once a head injury has occurred.
To lessen the risk of head injury, use the right safety equipment (such as hard hats, bicycle or motorcycle helmets, and seat belts).
Follow safety precautions at work and in sports and recreation. For example, do not dive into water if the water depth is unknown or if rocks may be present.
National Institute of Neurological Disorders and Stroke website. Traumatic brain injury: hope through research. www.ninds.nih.gov/Disorders/Patient-Caregiver-Education/Hope-Through-Research/Traumatic-Brain-Injury-Hope-Through. Updated April 24, 2020. Accessed November 3, 2020.
Shahlaie K, Zwienenberg-Lee M, Muizelaar JP. Clinical pathophysiology of traumatic brain injury. In: Winn HR, ed. Youmans and Winn Neurological Surgery. 7th ed. Philadelphia, PA: Elsevier; 2017:chap 346.
Wermers JD, Hutchison LH. Trauma. In: Coley BD, ed. Caffey's Pediatric Diagnostic Imaging. 13th ed. Philadelphia, PA: Elsevier; 2019:chap 39.