Metabolically Optimized, Non-cytotoxic Low Dose Weekly Decitabine/Venetoclax in MDS and AML

Status: Recruiting
Location: See all (3) locations...
Intervention Type: Drug
Study Type: Interventional
Study Phase: Phase 2
SUMMARY

Myeloid malignancies which include AML (acute myeloid leukemia) and MDS (myelodysplatic syndrome) are cancers of the bone marrow which lead to bone marrow failure. The bone marrow is the place or factory in the body where components of blood such as red cells, platelets and white cells are made. In bone marrow failure, the ability of the bone marrow to make these cells is decreased. The decreased bone marrow function is the result from abnormalities that develop in the malignant cells which prevent the normal maturation process by which bone marrow cells develop into red blood cells, white blood cells and platelets. The malignant cells in the bone marrow are not good at maturing to make the components of the blood that you need, they occupy space in the bone marrow and prevent the function of remaining normal bone marrow cells. DNA is a chemical substance within cells that stores information needed for cell growth and cell behavior. One approach to treating the malignant cells is to give chemotherapy which damages DNA within these cells and causes their death. Unfortunately, such therapy has side-effects, since even normal cells can be affected by the treatment. Decitabine is FDA approved for treatment of MDS and AML. Venetoclax is approved for AML in combination with Azacitidine for patients with AML or are over age 75 or unfit for chemotherapy. In this study, Decitabine and venetoclax will be administered using a low dose weekly schedule in an attempt to improve efficacy by decreasing the side effects often seen when these drugs are given at standard dosing.

Eligibility
Participation Requirements
Sex: All
Minimum Age: 18
Healthy Volunteers: f
View:

• Patient must have a diagnosis of MDS (myelodysplastic syndrome), AML (acute myeloid leukemia) or MDS/MPN (myelodysplastic/myeloproliferative neoplasms) with a histopathologic diagnosis confirmed by hematopathology review

• Indication for therapy with potential sensitivity to HMA (hypomethylating agents) therapy, defined as prior published evidence of response to HMA.

• Patients must be 18 years of age or older

• Patients must have an ECOG (Eastern Cooperative Oncology Group) performance status of ≥ 3

• Patients must have adequate end organ function defined as.

‣ AST (aspartate aminotransferase) and ALT (alanine transaminase) \< 4× the upper limit of normal (ULN)

⁃ Bilirubin ≤ 2× the ULN (upper limit of normal). If elevated bilirubin is due to impaired conjugation (e.g Gilbert's disease or concomitant medication) or disease related hemolysis, then direct bilirubin ≤ 1.5× the ULN.

⁃ As decitabine and venetoclax have little renal metabolism, and have proven safety even in dialysis patients, renal function with a creatinine clearance ≥30 mL/min or on dialysis is allowed.18

• Subjects must have the ability to understand and the willingness to sign a written informed consent document and complete study related procedures.

Locations
United States
California
University of California Davis Health (UC Davis Health)
RECRUITING
Sacramento
New York
Montefiore Medical Center
RECRUITING
The Bronx
White Plains Hospital
RECRUITING
White Plains
Contact Information
Primary
Mendel Goldfinger, MD
mgoldfin@montefiore.org
718-920-4826
Time Frame
Start Date: 2022-03-23
Estimated Completion Date: 2027-03
Participants
Target number of participants: 85
Treatments
Experimental: Decitabine/Venetoclax (Single Arm)
Administration: Decitabine is reconstituted with 5 ml sterile water to facilitate subcutaneous administration. Decitaboine is given by subcutaneous injection. Venetoclax is taken as a tablet prepared by patients pharmacy. Venetoclax is given at a dose of 400 mg po once per week concurrently with the Decitabine dose (+/- 1 day allowed ).
Related Therapeutic Areas
Sponsors
Collaborators: The V Foundation for Cancer Research
Leads: Montefiore Medical Center

This content was sourced from clinicaltrials.gov

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