Natural History and Long Term Clinical Assessments of All Forms of Neuronal Ceroid Lipofuscinoses - Capturing Key Symptoms and Disease Progression as Part of the Independent, International NCL DEM-CHILD Patient Database

Status: Recruiting
Location: See location...
Intervention Type: Other
Study Type: Observational
SUMMARY

This is an observational study that aims at assessing the natural history of NCL diseases as part of the international DEM-CHILD Database. 1. Patient data are collected from medical records, patient questionnaires and routine follow up clinical examinations with focus on assessing progression in key areas of disease such as motor, language, cognition, seizures, vision, and behavior. 2. A local biorepository of samples from genetically defined NCL patients will be established as well as a virtual biorepository within the DEM-CHILD DB to be able to easily localize international availability of patient samples.

Eligibility
Participation Requirements
Sex: All
Healthy Volunteers: f
View:

⁃ \- Patients with a confirmed molecular diagnosis of a form of NCL Disease

⁃ Additional inclusion criteria for Group/Cohort: CLN2 Disease - ERT (Brineura) Treated:

• Documented diagnosis of TPP1 deficiency

• Previous or current treatment with intracerebroventricular ERT with cerliponase alpha

• Patients that are currently participating in post-marketing studies will be allowed to participate.

Locations
Other Locations
Germany
University Medical Center Hamburg-Eppendorf
RECRUITING
Hamburg
Contact Information
Primary
Miriam Nickel, MD
m.nickel@uke.de
+4940741020440
Backup
Angela Schulz, MD, PhD
anschulz@uke.de
+4940741020440
Time Frame
Start Date: 2020-04-08
Estimated Completion Date: 2050-04-08
Participants
Target number of participants: 500
Treatments
CLN1 Disease, Haltia-Santavuori Disease
Patients with genetic mutations in the CLN1/PPT1 gene, causing a lysosomal enzyme deficiency of PPT1.
CLN2 Disease, Jansky-Bielschowsky Disease
Patients with genetic mutations in the CLN2/TPP1 gene, causing a lysosomal enzyme deficiency of TTP1.
CLN2 Disease - ERT (Brineura) treated
Patients with genetic mutations in the CLN2/TPP1 gene, causing a lysosomal enzyme deficiency of TTP1, previously and/or currently receiving enzyme-replacement therapy (ERT) with Cerliponase alpha (Brineura).
CLN3 Disease, Spielmeyer-Vogt-Sjögren-Batten Disease
Patients with genetic mutations in the CLN3 gene.
CLN4 disease, Parry disease
Patients with genetic mutations in the CLN4/DNAJC5 gene.
CLN5 Disease
Patients with genetic mutations in the CLN5 gene.
CLN6 Disease, Kufs Disease Type A
Patients with genetic mutations in the CLN6 gene.
CLN7 Disease
Patients with genetic mutations in the CLN7/MFSD8 gene.
CLN8 Disease
Patients with genetic mutations in the CLN8 gene.
CLN10 Disease
Patients with genetic mutations in the CLN10/CTSD gene, causing a lysosomal enzyme deficiency of Cathepsin D.
CLN11 Disease
Patients with genetic mutations in the CLN11/GRN gene.
CLN12 Disease
Patients with genetic mutations in the CLN12/ATP13A2 gene.
CLN13 Disease, Kufs Disease Type B
Patients with genetic mutations in the CLN13/CTSF gene, causing a lysosomal enzyme deficiency of Cathepsin F.
CLN14 Disease
Patients with genetic mutations in the CLN14/KCTD7 gene.
Sponsors
Leads: Universitätsklinikum Hamburg-Eppendorf

This content was sourced from clinicaltrials.gov

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