A Phase I/IIa Dose Escalation Study Evaluating the Safety and Efficacy of Autologous CD34+-enriched HSPCs Genetically Modified with Human Interferon-α2 in Patients with Glioblastoma Multiforme and Unmethylated MGMT Gene Promoter

Who is this study for? Patients with Glioblastoma
What treatments are being studied? Temferon
Status: Recruiting
Location: See all (3) locations...
Intervention Type: Drug
Study Type: Interventional
Study Phase: Phase 1/Phase 2
SUMMARY

This is a non-randomized, open label, phase I/IIa, dose-escalation study, involving a single injection of Temferon, an investigational advanced therapy consisting of autologous CD34+-enriched hematopoietic stem and progenitor cells exposed to transduction with a lentiviral vector driving myeloid specific interferon-alpha2 expression, which will be administered to up to 27 patients affected by GBM who have an unmethylated MGMT promoter. Part A will evaluate the safety and tolerability of 5 escalating doses of Temferon and 3 different conditioning regimens in up to 27 patients, following first line treatment.

Eligibility
Participation Requirements
Sex: All
Minimum Age: 18
Maximum Age: 70
Healthy Volunteers: f
View:

• Histologically confirmed, newly diagnosed supratentorial glioblastoma with unmethylated MGMT gene promoter.

• Patients have undergone complete or partial tumor resection.

• Able and willing to provide written informed consent and comply with the study protocol and procedures.

• Eligible for radiotherapy.

• Life expectancy of 6 months or more at Screening.

• Women of child-bearing potential enrolled in the study must have a negative pregnancy test at screening and agree to use acceptable methods of contraception during the trial.

• Men enrolled in the study with partners who are women of child-bearing potential, must be willing to use an acceptable barrier contraceptive method during the trial or have undergone successful vasectomy at least 6 months prior to entry into the study. Successful vasectomy needs to have been confirmed by semen analysis.

• Karnofsky performance score (KPS)≥70.

⁃ Additional inclusion criteria to be assessed within 20 days of Temferon administration:

• Adequate cardiac, renal, hepatic and pulmonary function as evidenced by:

• Left ventricular ejection fraction (LVEF) ≥ 45% by echo and normal electrocardiogram (ECG) or presence of abnormalities not significant for cardiac disease.

• Absence of severe pulmonary hypertension;

• Diffusing capacity of the lung for carbon monoxide (DLCO) \>50% and forced expiratory volume in 1 sec (FEV1) and forced expiratory vital capacity (FVC) \> 60% predicted (if non cooperative: pulse oximetry \> 95% in room air);

• Serum creatinine \< 2x upper limit normal and estimated glomerular filtration rate (eGFR) ≥ 30ml/min/1.73m\^2;

• Alkaline phosphatase (ALP), alanine aminotransferase (ALT) and/or aspartate aminotransferase (AST) ≤ 2.5 x upper limit of normal (ULN), and total bilirubin ≤ 2.0 mg/dl.

• Hemoglobin ≥10 g/dL, platelet count ≥100000/mm\^3, absolute neutrophil count \>1500/mm\^3.

Locations
Other Locations
Italy
Fondazione IRCCS Istituto Neurologico Carlo Besta
RECRUITING
Milan
Ospedale San Raffaele
ENROLLING_BY_INVITATION
Milan
Policlinico Universitario Fondazione Agostino Gemelli
NOT_YET_RECRUITING
Rome
Contact Information
Primary
Carlo Russo, MD
info-trial@genenta.com
+39 02 2643 3982
Backup
Stefania Mazzoleni, PhD
info-trial@genenta.com
+39 02 2643 3982
Time Frame
Start Date: 2019-03-05
Estimated Completion Date: 2025-12-09
Participants
Target number of participants: 27
Treatments
Experimental: Temferon
Autologous CD34+-enriched hematopoietic progenitor cells exposed in vitro to specific lentiviral vector encoding for the human interferon-alpha 2 gene. Its expression is tightly controlled by the human TIE2 enhancer/promoter sequence and by a post-transcriptional regulation layer represented by target miRNA sequences. This enables suppression of interferon-alpha2 expression in HSPCs, thereby further increasing the specificity of the delivery strategy for their Tie2 expressing myeloid cell progeny.
Sponsors
Leads: Genenta Science

This content was sourced from clinicaltrials.gov

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